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anti-Human IL34 Antibodies:
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Mouse (Murine) Monoclonal IL34 Primary Antibody for BR, FACS - ABIN2665836
Lin, Lee, Hestir, Leo, Huang, Bosch, Halenbeck, Wu, Zhou, Behrens, Hollenbaugh, Linnemann, Qin, Wong, Chu, Doberstein, Williams: Discovery of a cytokine and its receptor by functional screening of the extracellular proteome. in Science (New York, N.Y.) 2008
Show all 5 references for ABIN2665836
Mouse (Murine) Polyclonal IL34 Primary Antibody for BR, FACS - ABIN2660791
Chihara, Suzu, Hassan, Chutiwitoonchai, Hiyoshi, Motoyoshi, Kimura, Okada: IL-34 and M-CSF share the receptor Fms but are not identical in biological activity and signal activation. in Cell death and differentiation 2010
Show all 5 references for ABIN2660791
Human Monoclonal IL34 Primary Antibody for FACS - ABIN2660793
Eda, Shimada, Beidler, Monahan: Proinflammatory cytokines, IL-1β and TNF-α, induce expression of interleukin-34 mRNA via JNK- and p44/42 MAPK-NF-κB pathway but not p38 pathway in osteoblasts. in Rheumatology international 2011
Show all 5 references for ABIN2660793
Human Monoclonal IL34 Primary Antibody for ICC, FACS - ABIN969216
Slomiany, Baker, Elliott, Grossel: Changes in motility, gene expression and actin dynamics: Cdk6-induced cytoskeletal changes associated with differentiation in mouse astrocytes. in Journal of cellular biochemistry 2006
miR (show MLXIP Antibodies)-28-5p-IL-34-macrophage feedback loop modulates hepatocellular carcinoma metastasis
findings demonstrated that M-CSF (show CSF1 Antibodies) binds to IL-34; molecular docking studies predicted the formation of a heteromeric M-CSF (show CSF1 Antibodies)/IL-34 cytokine
These results suggest that IL-34, a novel osteoclastogenic cytokine, plays a role in rheumatoid arthritis-associated joint damage and is a potential biomarker for predicting subsequent radiographic progression in patients with RA.
IL-34 is expressed by human FOXP3 (show FOXP3 Antibodies)+CD45RCloCD8+ and CD4 (show CD4 Antibodies)+ Tregs and markedly inhibited alloreactive immune responses.
This paper provides evidence of alternative binding of IL-34 to chondroitin sulphates and syndecan-1 (show SDC1 Antibodies) at the cell surface that modulates M-CSFR (show CSF1R Antibodies) activation.
In vitro and in vivo experiments indicate that IL-34 expression is regulated by TNF-a (show TNF Antibodies) and IL-1b (show IL1B Antibodies) and that its overexpression is associated with an increase in osteosarcoma growth and metastasis.
the expression pattern of IL-34 in ileum and colon and suggest IL-34 as a new modulator of inflammation in inflammatory bowel disease
IL-34 is up-regulated in inflammatory bowel disease and suggest a role for this cytokine in sustaining the inflammatory responses in this disease
IL-34 is induced by IL-22 (show IL22 Antibodies) in the inflammatory cascade in response to IAV infection.
IL-34 expression in human gingival fibroblasts, stimulated by TNF-alpha (show TNF Antibodies) and IL-1beta (show IL1B Antibodies)
study concludes that Langerhans cells require IL-34 when residing in fully differentiated and anatomically intact skin epidermis, but rely on neutrophil-derived CSF1 (show CSF1 Antibodies) during inflammation
constitutive IL-34 expressed by skin keratinocytes might suppress resident macrophage responses to C. albicans colonisation by maintaining low levels TLR2 (show TLR2 Antibodies) and Dectin-1 (show CLEC7A Antibodies) expression by macrophages.
IL-34 protected blood-brain barrier integrity by restored expression levels of tight junction proteins, which were downregulated by pro-inflammatory cytokines.
IL-34-dependent, Mo-mediated, CSF-1 (show CSF1 Antibodies) nonredundant mechanisms promote persistent ischemia-incited AKI that worsens subsequent CKD.
Tumor necrosis factor-alpha (show TNF Antibodies) induces IL-34 expression via NF-kappaB (show NFKB1 Antibodies) in MC3T3-E1 osteoblastic cells.
These findings suggest that TGF-beta (show TGFB1 Antibodies) produced by IL-34-treated microglia is a negative regulator of microglial proliferation and enhances the neuroprotective property of microglia.
Differentiated signaling between IL-34 and CSF-1 (show CSF1 Antibodies) is likely achieved by the relative thermodynamic independence of IL-34 versus negative cooperativity of CSF-1 (show CSF1 Antibodies) at the CSF-1 receptor (show CSF1R Antibodies) recognition sites.
IL-34 as a nonredundant cytokine for the development of Langerhans cells during embryogenesis as well as for their homeostasis in the adult skin.
Interleukin-34 is a cytokine that promotes the differentiation and viability of monocytes and macrophages through the colony-stimulating factor-1 receptor (CSF1R\; MIM 164770) (Lin et al., 2008