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The protein encoded by CEND1 is a neuron-specific protein. Additionally we are shipping Cell Cycle Exit and Neuronal Differentiation 1 Proteins (6) and many more products for this protein.
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The detection of ALT, AFP (show AFP Antibodies), AFP (show AFP Antibodies)-L3, and GP73 (show GOLM1 Antibodies) has a certain guiding significance to predict the risk of hepatocellular carcinoma in hepatic cirrhosis patients
The mechanism of hepatocarcinogenesis and a promising blueprint for miR (show MLXIP Antibodies)-493-5p-GP73 (show GOLM1 Antibodies) axis-oriented treatment of HCC (show FAM126A Antibodies).
GP73 (show GOLM1 Antibodies) unglycosylation is associated with hepatocellular carcinoma cell motility and invasiveness.
The GOLPH2 (show GOLM1 Antibodies) is a novel marker for non-small-cell lung cancer.
Findings suggest that GP73 (show GOLM1 Antibodies) silencing through siRNA delivery may provide a low-toxicity therapy for the inhibition of tumor proliferation and metastasis.
Both gain- and loss-of-function studies determine that GOLM1 (show GOLM1 Antibodies) acts as a key oncogene (show RAB1A Antibodies) by promoting HCC (show FAM126A Antibodies) growth and metastasis.
A cytokine QTL at the NAA35 (show MAK10 Antibodies)-GOLM1 (show GOLM1 Antibodies) locus markedly modulated interleukin (IL)-6 (show IL6 Antibodies) production in response to multiple pathogens and was associated with susceptibility to candidemia.
knock-down of GP73 (show GOLM1 Antibodies) down-regulates HCV infection and replication in Huh7-MAVSR cells and primary human hepatocytes
Studies indicate that golgi membrane protein 1 (GOLM1 (show GOLM1 Antibodies)) may promote HCC (show FAM126A Antibodies) by regulation of epidermal growth factor receptor (EGFR (show EGFR Antibodies)) recycling, and suggest that therapeutic targeting of GOLM1 (show GOLM1 Antibodies) may be a potential strategy for combating hepatocellular carcinoma (HCC (show FAM126A Antibodies)) metastasis.
these studies revealed that GP73 (show GOLM1 Antibodies) promotes hepatitis C virus (HCV) secretion by directly mediating the interaction of ApoE (show APOE Antibodies) with HCV replication complex through binding with HCV NS5A.
data suggest that common reprogramming mechanisms exist driving the conversion of lineage-distant somatic cell types to neurons and reveal a critical role for CEND1 in NEUROG2 (show NEUROG2 Antibodies)-driven astrocytic reprogramming.
results suggest that functional interactions between BM88/Cend1, RanBPM and Dyrk1B (show DYRK1B Antibodies) affect the balance between cellular proliferation and differentiation in Neuro 2a cells
This study suggested that Cend1 is involved in Ahi1 (show AHI1 Antibodies)-associated hypothalamic neuronal differentiation in early development, giving us fresh insight into the mechanism behind the delayed development in Joubert syndrome.
Our results suggest that Cend1 is required for normal cerebellar development.
Transplantation of BM88/Cend1-overexpressing neural progenitor cells exerts beneficial effects on tissue regeneration by enhancing the number of generated neurons and restricting the formation of astroglial scar, in a mouse model of cortical brain injury.
BM88 is widely expressed in the embryonic CNS and PNS, in both nestin (show NES Antibodies)-positive neuroepithelial cells and post-mitotic betaIII-tubulin (show TUBB3 Antibodies) positive neurons.
Results implicate BM88 in the synchronization of cell cycle exit and differentiation of neuronal precursors in the developing nervous system.
BM88/Cend1 participates in cell cycle control and neuronal differentiation mechanisms during neonatal SVZ neurogenesis and becomes crucial for the transition from neuroblasts to mature neurons when reaching high levels
BM88 plays an essential role in regulating cell cycle exit and differentiation of Neuro 2a cells toward a neuronal phenotype
The protein encoded by this gene is a neuron-specific protein. The similar protein in pig enhances neuroblastoma cell differentiation in vitro and may be involved in neuronal differentiation in vivo. Multiple pseudogenes have been reported for this gene.
, cell cycle exit and neuronal differentiation protein 1
, cell cycle exit and neuronal differentiation 1