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Septin 5 antibody (N-Term)

The Rabbit Polyclonal anti-Septin 5 antibody has been validated for WB and IHC. It is suitable to detect Septin 5 in samples from Human.
Catalog No. ABIN2798741

Quick Overview for Septin 5 antibody (N-Term) (ABIN2798741)

Target

See all Septin 5 (SEPT5) Antibodies
Septin 5 (SEPT5)

Reactivity

  • 52
  • 27
  • 9
  • 2
Human

Host

  • 43
  • 9
Rabbit

Clonality

  • 43
  • 8
  • 1
Polyclonal

Conjugate

  • 29
  • 4
  • 4
  • 2
  • 1
  • 1
  • 1
  • 1
  • 1
  • 1
  • 1
  • 1
  • 1
  • 1
  • 1
  • 1
  • 1
This Septin 5 antibody is un-conjugated

Application

  • 49
  • 25
  • 24
  • 13
  • 13
  • 8
  • 7
  • 6
  • 5
  • 3
  • 3
  • 1
  • 1
  • 1
Western Blotting (WB), Immunohistochemistry (IHC)
  • Binding Specificity

    • 15
    • 9
    • 4
    • 4
    • 3
    • 2
    • 2
    • 1
    • 1
    AA 1-30, N-Term

    Purpose

    Rabbit Anti-Human SEPT5 (N-term) Antibody

    Immunogen

    This SEPT5 antibody is generated from rabbits immunized with a KLH conjugated synthetic peptide between 1-30 amino acids from the N-terminal region of human SEPT5.

    Isotype

    Ig Fraction
  • Application Notes

    Western Blot, Immunohistochemistry
    Recommended Dilutions
    WB: 1:1000, IHC: 1:10-50Western blot analysis of lysate from human brain tissue lysate, using SEPT5 Antibody (N-term) for immunohistochemistry. Clinical relevance has not been evaluated.

    Restrictions

    For Research Use only
  • Format

    Liquid

    Concentration

    0.5 mg/mL

    Storage

    4 °C,-20 °C

    Storage Comment

    2-8°C (short-term), -20°C (long-term)
  • Target

    Septin 5 (SEPT5)

    Alternative Name

    SEPT5

    Background

    Target Description: This gene is a member of the septin gene family of nucleotide binding proteins, originally described in yeast as cell division cycle regulatory proteins. Septins are highly conserved in yeast, Drosophila, and mouse and appear to regulate cytoskeletal organization. Disruption of septin function disturbs cytokinesis and results in large multinucleate or polyploid cells. This gene is mapped to 22q11, the region frequently deleted in DiGeorge and velocardiofacial syndromes. A translocation involving the MLL gene and this gene has also been reported in patients with acute myeloid leukemia. Alternative splicing results in multiple transcript variants. The presence of a non-consensus polyA signal (AACAAT) in this gene also results in read-through transcription into the downstream neighboring gene (GP1BB, platelet glycoprotein Ib), whereby larger, non-coding transcripts are produced. [provided by RefSeq].

    Gene Symbol: SEPT5

    Molecular Weight

    42777 Da

    Gene ID

    5413

    UniProt

    Q99719

    Pathways

    Synaptic Vesicle Exocytosis
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