FGF23 Protein (Myc-DYKDDDDK Tag)
Quick Overview for FGF23 Protein (Myc-DYKDDDDK Tag) (ABIN2721087)
Target
See all FGF23 ProteinsProtein Type
Origin
Source
Application
Purity
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Purification tag / Conjugate
- This FGF23 protein is labelled with Myc-DYKDDDDK Tag.
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Characteristics
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- Recombinant human FGF23 protein expressed in HEK293 cells.
- Produced with end-sequenced ORF clone
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Fibroblast Growth Factor 23 (FGF23) (AA 25-251) protein (His tag)
FGF23 Origin: Human Host: Escherichia coli (E. coli) Recombinant Greater than 90 % as determined by SDS-PAGE. WB, ELISA
Fibroblast Growth Factor 23 (FGF23) (AA 25-251) protein (His-IF2DI Tag)FGF23 Origin: Mouse Host: Escherichia coli (E. coli) Recombinant Greater than 90 % as determined by SDS-PAGE. WB, ELISA
Fibroblast Growth Factor 23 (FGF23) (AA 1-251) protein (GST tag)FGF23 Origin: Human Host: Wheat germ Recombinant AP, AA, ELISA, WB
Fibroblast Growth Factor 23 (FGF23) (AA 25-251) (Active) protein (His tag)FGF23 Origin: Mouse Host: Escherichia coli (E. coli) Recombinant > 95 % AcA, CC Active
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Application Notes
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Recombinant human proteins can be used for:
Native antigens for optimized antibody production
Positive controls in ELISA and other antibody assays -
Comment
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The tag is located at the C-terminal.
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Restrictions
- For Research Use only
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Concentration
- 50 μg/mL
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Buffer
- 25 mM Tris.HCl, pH 7.3, 100 mM glycine, 10 % glycerol.
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Storage
- -80 °C
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Storage Comment
- Store at -80°C. Thaw on ice, aliquot to individual single-use tubes, and then re-freeze immediately. Only 2-3 freeze thaw cycles are recommended.
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- FGF23 (Fibroblast Growth Factor 23 (FGF23))
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Alternative Name
- Fgf23
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Background
- This gene encodes a member of the fibroblast growth factor family of proteins, which possess broad mitogenic and cell survival activities and are involved in a variety of biological processes. The product of this gene regulates phosphate homeostasis and transport in the kidney. The full-length, functional protein may be deactivated via cleavage into N-terminal and C-terminal chains. Mutation of this cleavage site causes autosomal dominant hypophosphatemic rickets (ADHR). Mutations in this gene are also associated with hyperphosphatemic familial tumoral calcinosis (HFTC).
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Molecular Weight
- 25.3 kDa
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NCBI Accession
- NP_065689
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Pathways
- RTK Signaling, Fc-epsilon Receptor Signaling Pathway, EGFR Signaling Pathway, Neurotrophin Signaling Pathway, Negative Regulation of Hormone Secretion
Target
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