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The CTX (see VSIG2, MIM 606011) family of proteins, including ASAM, are type I transmembrane proteins within the Ig superfamily that localize to junctional complexes between endothelial and epithelial cells and may play a role in cell-cell adhesion (Raschperger et al., 2004 [PubMed 14573622]).[supplied by OMIM, Mar 2008].. Additionally we are shipping CLMP Proteins (16) and CLMP Kits (7) and many more products for this protein.
Showing 10 out of 59 products:
Human Polyclonal CLMP Primary Antibody for EIA, WB - ABIN950225
Kawabata, Tashiro, Sakurai, Osada, Kusuda, Hayakawa, Yamanishi, Mizuguchi: Positive and negative regulation of adenovirus infection by CAR-like soluble protein, CLSP. in Gene therapy 2007
Show all 5 references for ABIN950225
Human Polyclonal CLMP Primary Antibody for IHC (p), WB - ABIN656961
Eguchi, Wada, Hida, Zhang, Matsuoka, Baba, Hashimoto, Shikata, Ogawa, Makino: Identification of adipocyte adhesion molecule (ACAM), a novel CTX gene family, implicated in adipocyte maturation and development of obesity. in The Biochemical journal 2005
Show all 3 references for ABIN656961
Human Polyclonal CLMP Primary Antibody for ELISA, WB - ABIN4281764
Raschperger, Engstrom, Pettersson, Fuxe: CLMP, a novel member of the CTX family and a new component of epithelial tight junctions. in The Journal of biological chemistry 2003
The key processes involved in intestinal epithelial development appear to be unaffected by wild type-CLMP or mutant-CLMP.
Coxsackievirus and adenovirus receptor (show CXADR Antibodies) gene expression is induced in esophageal cancer cells by the HDAC (show HDAC3 Antibodies) inhibitor trichostatin A.
The PDZ1 and PDZ3 domains of MAGI-1 (show MAGI1 Antibodies) regulate the eight-exon isoform of the CXADR-like membrane protein.
Loss-of-function mutations in CLMP cause congenital short bowel syndrome in human beings, likely by interfering with tight-junction formation, which disrupts intestinal development.
CLMP is a novel cell-cell adhesion molecule (show PCDHB16 Antibodies) and a new component of epithelial tight junctions.
We identified ACAM (adipocyte adhesion molecule), a novel homologue of the CTX (cortical thymocyte marker (show VSIG1 Antibodies) in Xenopus) gene family, which may be the critical adhesion molecule (show NCAM1 Antibodies) in adipocyte differentiation and development of obesity.
ASAM, IGSF11 (show IGSF11 Antibodies), CXADR (show CXADR Antibodies) and ESAM (show ESAM Antibodies) are type I transmembrane proteins and members of the same IGSF superfamily.
Expression of CLMP, a novel tight junction protein (show OCLN Antibodies), is mediated via the interaction of GATA (show QRSL1 Antibodies) with the Kruppel family proteins, KLF4 (show KLF4 Antibodies) and Sp1 (show SP1 Antibodies), in mouse TM4 (show TPM4 Antibodies) Sertoli cells.
TNFalpha (show TNF Antibodies)-mediated mRNA degradation of the CLMP gene is controlled by TTP (show ZFP36 Antibodies) through the JNK (show MAPK8 Antibodies) signalling cascade
The CTX (see VSIG2, MIM 606011) family of proteins, including ASAM, are type I transmembrane proteins within the Ig superfamily that localize to junctional complexes between endothelial and epithelial cells and may play a role in cell-cell adhesion (Raschperger et al., 2004
CAR-like membrane protein
, adipocyte adhesion molecule
, adipocyte-specific adhesion molecule
, coxsackie- and adenovirus receptor-like membrane protein
, visceral adipose tissue-specific transmembrane protein OL-16
, adipocyte-specific protein 5