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The protein encoded by DBP is a member of the PAR bZIP transcription factor family and binds to specific sequences in the promoters of several genes, such as albumin, CYP2A4, and CYP2A5. Additionally we are shipping DBP Kits (11) and DBP Proteins (3) and many more products for this protein.
Showing 10 out of 19 products:
Cow (Bovine) Polyclonal DBP Primary Antibody for IHC, WB - ABIN2779501
Smith, Tachibana, Pohl, Lee, Thanarajasingam, Portier, Ueki, Ramaswamy, Billings, Mohrenweiser, Louis, Jenkins: A transcript map of the chromosome 19q-arm glioma tumor suppressor region. in Genomics 2000
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Cow (Bovine) Polyclonal DBP Primary Antibody for IHC, WB - ABIN2777367
Hamamura, Matsunaga, Ikeda, Kondo, Ikeyama, Tokushige, Itcho, Furuichi, Yoshida, Matsuda, Yasuda, Doi, Yokota, Amamoto, Aramaki, Irino, Koyanagi, Ohdo: Alterations of Hepatic Metabolism in Chronic Kidney Disease via D-box-binding Protein Aggravate the Renal Dysfunction. in The Journal of biological chemistry 2016
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PI3K signaling plays a modulatory role in the regulation of the transcriptional rhythm of the Dbp (show GC Antibodies) gene by targeting BMAL1 (show ARNTL Antibodies) and CLOCK.
constitutive Dbp (show GC Antibodies) expression plays an important role in mediating hepatic dysfunction under chronic kidney disease. DBP (show GC Antibodies) was significantly decreased in 5/6Nx model mice.
Dbp (show GC Antibodies) plays a well-established role in circadian rhythms.
Expressions of Dbp (show GC Antibodies) and Arnt (show ARNT Antibodies) were dampened in the islets of a diabetic model mouse.
BMAL1 (show ARNTL Antibodies) and CLOCK may act as Kamikaze activators, in that they are rapidly degraded once bound to Dbp (show GC Antibodies) chromatin.
In Ccdc80 (show CCD80 Antibodies)(-/-) mice, expression of the core clock member Arntl/Bmal1 (show ARNTL Antibodies) was reduced whereas that of the oscillating transcription factors Dbp (show GC Antibodies) and Tef (show TEF Antibodies) was increased in all tissues examined.
The daily mRNA expression profiles of the clock and clock-controlled genes Clock, Bmal1 (show ARNTL Antibodies), Cry1 (show CRY1 Antibodies), Per1 (show PER1 Antibodies), Per2 (show PER2 Antibodies), and Dbp (show GC Antibodies) were substantially dampened in the liver and adipose tissue of 10-wk-old ob/ob mice.
we demonstrate that D site albumin promoter binding protein/thyrotroph embryonic factor (show TEF Antibodies)/hepatic leukemia factor (show HLF Antibodies) triple knockout mice develop cardiac hypertrophy and left ventricular dysfunction associated with a low blood pressure
CRY1 (show CRY1 Antibodies) binding to the Dbp (show GC Antibodies) promoter region delayed BMAL1 (show ARNTL Antibodies) and CLOCK-mediated transcription of Dbp (show GC Antibodies) compared with Rev-Erbalpha (show NR1D1 Antibodies)
mice deficient for all three PAR (show AFG3L2 Antibodies) bZip proteins (DBP (show GC Antibodies),HLF (show HLF Antibodies),& TEF (show TEF Antibodies)) are highly susceptible to generalized spontaneous and audiogenic epilepsies, frequently lethal. Pyridoxal kinase (show PDXK Antibodies) is a target gene of PAR (show AFG3L2 Antibodies) bZip proteins in both liver and brain
Data suggest that, while low circulating levels of DBP (show GC Antibodies) contribute to low circulating levels of 25-hydroxyvitamin D in patients with PHPT (primary hyperparathyroidism), low DBP (show GC Antibodies) alone is not responsible for the hypovitaminosis D observed in these patients; vitamin D metabolism is likely to be generally disturbed in PHPT. [EDITORIAL]
The interaction between vitamin D status, as measured by circulating 25(OH)D and DBP (show GC Antibodies) rs2282679 genotypes, modified the association between 25(OH)D and BMD (show BEST1 Antibodies) and bone markers.
the expression of DBP (show GC Antibodies) and MAPK (show MAPK1 Antibodies) in epilepsy patients was upregulated, suggesting a possible pathogenetic role in IE.
Increased circulating levels of vitamin D binding protein (show GC Antibodies) in multiple sclerosis patients.
Results show that APOE (show APOE Antibodies), DBP (show GC Antibodies) and AGT (show AGXT Antibodies) identified were associated with survival outcomes in metastatic colorectal cancer patients treated with chemotherapy and bevacizumab.
DBP (show GC Antibodies) strictly inhibited the production of 1alpha,25-dihydroxyvitamin D3 and 25-hydroxyvitamin D3-induced T cell responses.
25(OH)D2 half-life was shorter than 25(OH)D3 half-life, and half-lives were affected by DBP (show GC Antibodies) concentration and genotype.
Lean individuals exhibited significant (P < 0.05) temporal changes of core clock (PER1 (show PER1 Antibodies), PER2 (show PER2 Antibodies), PER3, CRY2 (show CRY2 Antibodies), BMAL1 (show ARNTL Antibodies), and DBP (show GC Antibodies)) and metabolic (REVERBalpha (show NR1D1 Antibodies), RIP140 (show NRIP1 Antibodies), and PGC1alpha) genes.
These results provide a direct evidence of cross-talk among the structural domains of DBP (show GC Antibodies).
Data show that vitamin D-binding protein (DBP (show GC Antibodies)) is elevated in the CSF (show CSF2 Antibodies) of temporal lobe epilepsy patients.
The protein encoded by this gene is a member of the PAR bZIP transcription factor family and binds to specific sequences in the promoters of several genes, such as albumin, CYP2A4, and CYP2A5. The encoded protein can bind DNA as a homo- or heterodimer and is involved in the regulation of some circadian rhythym genes.
D site of albumin promoter (albumin D-box) binding protein
, D site of albumin promoter binding protein
, D site-binding protein
, albumin D box-binding protein
, albumin D-element-binding protein
, tax-responsive enhancer element-binding protein 302
, D site albumin promoter binding protein
, d site albumin promoter-binding protein 1
, albumin D-element binding protein