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ICAM1 encodes a cell surface glycoprotein which is typically expressed on endothelial cells and cells of the immune system. Additionally we are shipping ICAM-1 Kits (140) and ICAM-1 Proteins (69) and many more products for this protein.
Showing 10 out of 970 products:
Rat (Rattus) Monoclonal ICAM-1 Primary Antibody for FACS, IHC (fro) - ABIN120500
Tamatani, Miyasaka et al.: Identification of monoclonal antibodies reactive with the rat homolog of ICAM-1, and evidence for a differential involvement of ICAM-1 in the adherence of resting versus activated lymphocytes to high ... in International immunology 1991
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Rat (Rattus) Monoclonal ICAM-1 Primary Antibody for Func, FACS - ABIN182014
Tamatani, Kotani, Tanaka, Miyasaka: Molecular mechanisms underlying lymphocyte recirculation. II. Differential regulation of LFA-1 in the interaction between lymphocytes and high endothelial cells. in European journal of immunology 1991
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Rat (Rattus) Monoclonal ICAM-1 Primary Antibody for FACS - ABIN120497
Neto, Oliveira: Nanoscillator and gun under temperature effect. in Journal of nanoscience and nanotechnology 2010
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Human Monoclonal ICAM-1 Primary Antibody for FACS, IHC (fro) - ABIN111935
Albelda, Buck: Integrins and other cell adhesion molecules. in FASEB journal : official publication of the Federation of American Societies for Experimental Biology 1990
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Human Monoclonal ICAM-1 Primary Antibody for FACS, IHC (fro) - ABIN111934
Freudenthal, Steinman: The distinct surface of human blood dendritic cells, as observed after an improved isolation method. in Proceedings of the National Academy of Sciences of the United States of America 1990
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Human Monoclonal ICAM-1 Primary Antibody for FACS - ABIN1106462
Champagne, Tremblay, Cantin, St Pierre: Proteolytic cleavage of ICAM-1 by human neutrophil elastase. in Journal of immunology (Baltimore, Md. : 1950) 1998
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Human Monoclonal ICAM-1 Primary Antibody for FACS, IHC (fro) - ABIN118785
Dransfield, Cabañas, Barrett, Hogg: Interaction of leukocyte integrins with ligand is necessary but not sufficient for function. in The Journal of cell biology 1992
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Human Monoclonal ICAM-1 Primary Antibody for FACS - ABIN1106463
Vermot-Desroches, Marchand, Roy, Wijdenes: Heterogeneity of antigen expression among human umbilical cord vascular endothelial cells: identification of cell subsets by co-expression of haemopoietic antigens. in Immunology letters 1996
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Human Monoclonal ICAM-1 Primary Antibody for FACS, IP - ABIN1106457
Spirig, van Kooten, Obregon, Nicod, Daha, Rieben: The complement inhibitor low molecular weight dextran sulfate prevents TLR4-induced phenotypic and functional maturation of human dendritic cells. in Journal of immunology (Baltimore, Md. : 1950) 2008
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Human Polyclonal ICAM-1 Primary Antibody for ELISA, WB - ABIN1532900
Simmons, Makgoba, Seed: ICAM, an adhesion ligand of LFA-1, is homologous to the neural cell adhesion molecule NCAM. in Nature 1988
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This study investigated the possibility of K469E (rs5498) and G241R (rs1799969) polymorphisms, in ICAM-1 gene, and G634C (rs1800796), in IL-6 (show IL6 Antibodies) gene, being associated with the occurrence of endometriosis . All individuals were wild homozygotes for G241R polymorphism. This study suggests that polymorphisms K469E, G241R, and G634C are not associated with increased susceptibility to endometriosis
ICAM-1 expression was significantly lower in adult T-cell leukemia/lymphoma patients.
Activin A (show INHBA Antibodies) and preeclamptic serum upregulate ET-1 (show EDN1 Antibodies), ICAM-1, and VCAM-1 (show VCAM1 Antibodies) in human umbilical vein endothelial cells, consistent with activin A (show INHBA Antibodies) contributing to the pathophysiology of preeclampsia.
Statistically significant associations were identified between the chronic periodontal patients and the controls in the gene polymorphisms of rs5498 ICAM-1 (P=0.007) and rs1041163 VCAM-1 (show VCAM1 Antibodies) (P=0.029). The distribution of rs5498 (P=0.029) and rs1041163 (P=0.049) differed significantly across the mild, moderate, and severe groups of periodontitis.
Blood soluble ICAM-1 levels are decreased significantly in patients with community-acquired pneumonia.
MiR (show MLXIP Antibodies)-146a regulates inflammatory macrophage infiltration in Polymyositis/Dermatomyositis by targeting TRAF6 (show TRAF6 Antibodies) and affecting the IL-17 (show IL17A Antibodies)/ICAM-1 pathway.
The intercellular adhesion molecule-1 K469E polymorphism is associated with susceptibility to vasculitis under the heterozygote model, and the intercellular adhesion molecule-1 G241R polymorphism is associated with vasculitis in Europeans, Behcet's disease, and rheumatoid arthritis.
Increased risk of colorectal cancer associated with a polymorphism in the ICAM-1 gene in a German population.
The results suggest that the genetic polymorphism in ICAM-1 rs5498 single nucleotide polymorphisms with genotype AG and GG is associated with hepatocellular carcinoma risk among smokers.
study provides review of the Genome Wide Association (GWA) studies and summarizes its understanding of functional Single Nucleotide Polymorphism (SNP's) of ICAM-1 clinical association
ICAM1 regulates pathological cardiac remodeling by mediating proinflammatory leukocyte infiltration in the left ventricle and cardiac fibrosis and dysfunction.
Its antiatherogenic impact might be associated with a suppressed adhesion to the endothelium due to down-regulation of endothelial ICAM-1 expression.
Luteolin may inhibit tumor angiogenesis and tumor cell proliferation by down-regulation of LFA- 3 and PCNA (show PCNA Antibodies) and up-regulation of ICAM-1 in tumor tissue of tumor-bearing mice, thereby achieving its anti-tumor effect.
our results further highlight the pleiotropic role of ICAM-1 in T-cell-dependent immune responses, with a major role in Treg cell development and suppressive function.
the present results shed light on regulation of expression and function of ICAM-1 on neutrophils and identify it as an additional regulator of neutrophil effector responses in host defense.
we identified intracellular adhesion molecule (ICAM)-1, through fibrinogen binding, as a previously unreported mediator of platelet-monocyte interactions
miR (show MLXIP Antibodies)-222 was transported into recipient endothelial cells by endothelial microparticles and functionally regulated expression of its target protein ICAM-1 in vitro and in vivo.
Timely blockade of ICAM-1.LFA-1 (show ITGAL Antibodies) interaction prevents disease onset in a mouse model of emphysema.
MRTF-A (show MKL1 Antibodies)/B depletion results in an increase in the cell surface expression of ICAM-1 and interactions between HAoECs and leukocytes
neutrophils are reliant on endothelial activation for adhesion, initially via E-selectin (show SELE Antibodies) and subsequently, by integrin-mediated binding to ICAM-1 and VCAM-1 (show VCAM1 Antibodies), combined with CXCL8 (show IL8 Antibodies)-dependent chemotaxis.
ADMA has potent adverse effects on cell proliferation, intracellular ROS (show ROS1 Antibodies) generation, cell permeability, levels of ICAM-1, and the tight-junction protein occludin (show OCLN Antibodies)
Neutrophil lung infiltrations in porcine reproductive and respiratory syndrome virus infection infected animals is both ICAM-1 dependent and independent.
Chitosan oligosaccharides downregulate the expression of E-selectin (show SELE Antibodies) and ICAM-1 by inhibiting the phosphorylation of Mitogen-Activated Protein Kinases and the activation of NF-kappaB (show NFKB1 Antibodies) in lipopolysaccharides treated porcine iliac artery endothelial cells.
Data show that all five molecules, BNP (show BNC2 Antibodies), ICAM-1, TNF-alpha (show TNF Antibodies), VCAM-1 (show VCAM1 Antibodies) and IL-6 (show IL6 Antibodies), quickly and reliably signaled adverse interactions.
Altered shear stress stimulates upregulation of endothelial VCAM-1 (show VCAM1 Antibodies) and ICAM-1 in a BMP-4 (show BMP4 Antibodies)- and TGF-beta1 (show TGFB1 Antibodies)-dependent pathway.
ICAM1 and IL10 (show IL10 Antibodies) were upregulated in ventilator-induced lung injury. Nuclear transcription factor AP-1 (show JUN Antibodies) may be responsible for this upregulation.
Hepatocellular glycogen (show GYS1 Antibodies) decreases the expression of ICAM-1 mRNA of hepatic stellate cells.
This gene encodes a cell surface glycoprotein which is typically expressed on endothelial cells and cells of the immune system. It binds to integrins of type CD11a / CD18, or CD11b / CD18 and is also exploited by Rhinovirus as a receptor.
intercellular adhesion molecule 1
, intercellular adhesion molecule 1-like
, cell surface glycoprotein P3.58
, intercellular adhesion molecule 1 (CD54), human rhinovirus receptor
, major group rhinovirus receptor
, leukocyte adhesion molecule