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ICAM1 encodes a cell surface glycoprotein which is typically expressed on endothelial cells and cells of the immune system. Additionally we are shipping ICAM-1 Kits (150) and ICAM-1 Proteins (59) and many more products for this protein.
Showing 10 out of 788 products:
Rat (Rattus) Monoclonal ICAM-1 Primary Antibody for FACS, IHC (fro) - ABIN120500
Tamatani, Miyasaka et al.: Identification of monoclonal antibodies reactive with the rat homolog of ICAM-1, and evidence for a differential involvement of ICAM-1 in the adherence of resting versus activated lymphocytes to high ... in International immunology 1991
Show all 11 references for ABIN120500
Rat (Rattus) Monoclonal ICAM-1 Primary Antibody for Func, FACS - ABIN182014
Tamatani, Kotani, Tanaka, Miyasaka: Molecular mechanisms underlying lymphocyte recirculation. II. Differential regulation of LFA-1 in the interaction between lymphocytes and high endothelial cells. in European journal of immunology 1991
Show all 10 references for ABIN182014
Rat (Rattus) Monoclonal ICAM-1 Primary Antibody for FACS - ABIN120497
Neto, Oliveira: Nanoscillator and gun under temperature effect. in Journal of nanoscience and nanotechnology 2010
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Human Monoclonal ICAM-1 Primary Antibody for FACS, IHC (fro) - ABIN111935
Albelda, Buck: Integrins and other cell adhesion molecules. in FASEB journal : official publication of the Federation of American Societies for Experimental Biology 1990
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Human Monoclonal ICAM-1 Primary Antibody for FACS, IHC (fro) - ABIN111934
Freudenthal, Steinman: The distinct surface of human blood dendritic cells, as observed after an improved isolation method. in Proceedings of the National Academy of Sciences of the United States of America 1990
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Human Monoclonal ICAM-1 Primary Antibody for FACS - ABIN1106462
Champagne, Tremblay, Cantin, St Pierre: Proteolytic cleavage of ICAM-1 by human neutrophil elastase. in Journal of immunology (Baltimore, Md. : 1950) 1998
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Human Monoclonal ICAM-1 Primary Antibody for FACS, IHC (fro) - ABIN118785
Dransfield, Cabañas, Barrett, Hogg: Interaction of leukocyte integrins with ligand is necessary but not sufficient for function. in The Journal of cell biology 1992
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Human Monoclonal ICAM-1 Primary Antibody for FACS - ABIN1106463
Vermot-Desroches, Marchand, Roy, Wijdenes: Heterogeneity of antigen expression among human umbilical cord vascular endothelial cells: identification of cell subsets by co-expression of haemopoietic antigens. in Immunology letters 1996
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Human Polyclonal ICAM-1 Primary Antibody for ELISA, WB - ABIN1532900
Simmons, Makgoba, Seed: ICAM, an adhesion ligand of LFA-1, is homologous to the neural cell adhesion molecule NCAM. in Nature 1988
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Human Monoclonal ICAM-1 Primary Antibody for FACS, IP - ABIN1106457
Spirig, van Kooten, Obregon, Nicod, Daha, Rieben: The complement inhibitor low molecular weight dextran sulfate prevents TLR4-induced phenotypic and functional maturation of human dendritic cells. in Journal of immunology (Baltimore, Md. : 1950) 2008
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Our results also supported the hypothesis that the KK genotype at the K469E locus in ICAM-1 is a risk factor for AIS (show AR Antibodies).
genetic association study in Malaysian population: Data suggest that an SNP in ICAM1 [rs5498, K469E(A/G)] is associated with diabetic nephropathy (DN) in subjects with type 2 diabetes in the population studied; plasma ICAM1 is up-regulated in DN.
The effect of IL-35 on electrical impedance reflecting tissue integrity, the surface expression of ICAM-1 and mRNA expression of IL-32 (show IL32 Antibodies), as well as apoptosis in human primary aortic smooth muscle cells (Ao-SMCs) was investigated
ABL (show ABL1 Antibodies)-N administration induced apoptosis of PC3 (show PCSK1 Antibodies) cells in a dose-dependent manner, along with the enhanced activity of caspases and increased Bax (show BAX Antibodies)/Bcl-2 (show BCL2 Antibodies) ratio. Expression of KLF5 (show KLF5 Antibodies), Stat5b (show STAT5B Antibodies) and ICAM-1 was significantly downregulated in PC3 (show PCSK1 Antibodies) cells.
Contact-dependent lung adenocarcinoma aggregate dispersion by M2a macrophages occurs via ICAM-1 and beta2 integrin interactions.
Lipopolysaccharides up-regulate ICAM-1 expression in breast cancer cells via a MyD88 (show MYD88 Antibodies)-BLT2 (show LTB4R2 Antibodies)-ERK (show EPHB2 Antibodies)-linked signaling cascade.
The genotypic distributions of ICAM-1 (K469E) were significantly different between patients and controls.
ICAM1 and CD44 (show CD44 Antibodies) could have a compensation effect on maintaining the stemness characteristics of esophageal squamous cell carcinoma.
expression levels of ICAM-1, NF-kappaB (show NFKB1 Antibodies), and MCP-1 (show CCL2 Antibodies) in patients are significantly elevated, suggesting an enhanced chronic inflammatory response and a significant correlation between inflammatory factors and the pathogenesis of cerebral aneurysm
Identify the selectins ICAM-1 and VCAM-1 (show VCAM1 Antibodies) as strong and independent predictors of all-cause and cardiovascular mortality in patients with stable carotid atherosclerosis.
Luteolin may inhibit tumor angiogenesis and tumor cell proliferation by down-regulation of LFA- 3 and PCNA (show PCNA Antibodies) and up-regulation of ICAM-1 in tumor tissue of tumor-bearing mice, thereby achieving its anti-tumor effect.
our results further highlight the pleiotropic role of ICAM-1 in T-cell-dependent immune responses, with a major role in Treg cell development and suppressive function.
the present results shed light on regulation of expression and function of ICAM-1 on neutrophils and identify it as an additional regulator of neutrophil effector responses in host defense.
we identified intracellular adhesion molecule (ICAM)-1, through fibrinogen binding, as a previously unreported mediator of platelet-monocyte interactions
miR (show MLXIP Antibodies)-222 was transported into recipient endothelial cells by endothelial microparticles and functionally regulated expression of its target protein ICAM-1 in vitro and in vivo.
Timely blockade of ICAM-1.LFA-1 (show ITGAL Antibodies) interaction prevents disease onset in a mouse model of emphysema.
MRTF-A (show MKL1 Antibodies)/B depletion results in an increase in the cell surface expression of ICAM-1 and interactions between HAoECs and leukocytes
neutrophils are reliant on endothelial activation for adhesion, initially via E-selectin (show SELE Antibodies) and subsequently, by integrin-mediated binding to ICAM-1 and VCAM-1 (show VCAM1 Antibodies), combined with CXCL8 (show IL8 Antibodies)-dependent chemotaxis.
Inhibition of the P2RX7 (show P2RX7 Antibodies) pathway not only decreased endothelial ICAM-1 expression and leukocyte adhesion but also prevented microglia overactivation, reduced brain injury, and consequently doubled the early survival of septic mice.
ICAM-1 in the tumor microenvironment, via restraining efferocytosis of apoptotic tumor cells, can block M2 macrophage polarization through regulation of PI3K/AKT (show AKT1 Antibodies) activation, which leads to prevention of tumor metastasis.
ADMA has potent adverse effects on cell proliferation, intracellular ROS (show ROS1 Antibodies) generation, cell permeability, levels of ICAM-1, and the tight-junction protein occludin (show OCLN Antibodies)
Neutrophil lung infiltrations in porcine reproductive and respiratory syndrome virus infection infected animals is both ICAM-1 dependent and independent.
Chitosan oligosaccharides downregulate the expression of E-selectin (show SELE Antibodies) and ICAM-1 by inhibiting the phosphorylation of Mitogen-Activated Protein Kinases and the activation of NF-kappaB (show NFKB1 Antibodies) in lipopolysaccharides treated porcine iliac artery endothelial cells.
Data show that all five molecules, BNP (show BNC2 Antibodies), ICAM-1, TNF-alpha (show TNF Antibodies), VCAM-1 (show VCAM1 Antibodies) and IL-6 (show IL6 Antibodies), quickly and reliably signaled adverse interactions.
Altered shear stress stimulates upregulation of endothelial VCAM-1 (show VCAM1 Antibodies) and ICAM-1 in a BMP-4 (show BMP4 Antibodies)- and TGF-beta1 (show TGFB1 Antibodies)-dependent pathway.
ICAM1 and IL10 (show IL10 Antibodies) were upregulated in ventilator-induced lung injury. Nuclear transcription factor AP-1 (show JUN Antibodies) may be responsible for this upregulation.
Hepatocellular glycogen (show GYS1 Antibodies) decreases the expression of ICAM-1 mRNA of hepatic stellate cells.
This gene encodes a cell surface glycoprotein which is typically expressed on endothelial cells and cells of the immune system. It binds to integrins of type CD11a / CD18, or CD11b / CD18 and is also exploited by Rhinovirus as a receptor.
intercellular adhesion molecule 1
, intercellular adhesion molecule 1-like
, cell surface glycoprotein P3.58
, intercellular adhesion molecule 1 (CD54), human rhinovirus receptor
, major group rhinovirus receptor
, leukocyte adhesion molecule