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ICAM1 encodes a cell surface glycoprotein which is typically expressed on endothelial cells and cells of the immune system. Additionally we are shipping ICAM-1 Kits (136) and ICAM-1 Proteins (60) and many more products for this protein.
Showing 10 out of 955 products:
Rat (Rattus) Monoclonal ICAM-1 Primary Antibody for FACS, IHC (fro) - ABIN120500
Tamatani, Miyasaka et al.: Identification of monoclonal antibodies reactive with the rat homolog of ICAM-1, and evidence for a differential involvement of ICAM-1 in the adherence of resting versus activated lymphocytes to high ... in International immunology 1991
Show all 11 references for ABIN120500
Rat (Rattus) Monoclonal ICAM-1 Primary Antibody for Func, FACS - ABIN182014
Tamatani, Kotani, Tanaka, Miyasaka: Molecular mechanisms underlying lymphocyte recirculation. II. Differential regulation of LFA-1 in the interaction between lymphocytes and high endothelial cells. in European journal of immunology 1991
Show all 10 references for ABIN182014
Rat (Rattus) Monoclonal ICAM-1 Primary Antibody for FACS - ABIN120497
Neto, Oliveira: Nanoscillator and gun under temperature effect. in Journal of nanoscience and nanotechnology 2010
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Human Monoclonal ICAM-1 Primary Antibody for FACS, IHC (fro) - ABIN111935
Albelda, Buck: Integrins and other cell adhesion molecules. in FASEB journal : official publication of the Federation of American Societies for Experimental Biology 1990
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Human Monoclonal ICAM-1 Primary Antibody for FACS, IHC (fro) - ABIN111934
Freudenthal, Steinman: The distinct surface of human blood dendritic cells, as observed after an improved isolation method. in Proceedings of the National Academy of Sciences of the United States of America 1990
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Human Monoclonal ICAM-1 Primary Antibody for FACS - ABIN1106462
Champagne, Tremblay, Cantin, St Pierre: Proteolytic cleavage of ICAM-1 by human neutrophil elastase. in Journal of immunology (Baltimore, Md. : 1950) 1998
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Human Monoclonal ICAM-1 Primary Antibody for FACS, IHC (fro) - ABIN118785
Dransfield, Cabañas, Barrett, Hogg: Interaction of leukocyte integrins with ligand is necessary but not sufficient for function. in The Journal of cell biology 1992
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Human Monoclonal ICAM-1 Primary Antibody for FACS - ABIN1106463
Vermot-Desroches, Marchand, Roy, Wijdenes: Heterogeneity of antigen expression among human umbilical cord vascular endothelial cells: identification of cell subsets by co-expression of haemopoietic antigens. in Immunology letters 1996
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Human Monoclonal ICAM-1 Primary Antibody for FACS, IP - ABIN1106457
Spirig, van Kooten, Obregon, Nicod, Daha, Rieben: The complement inhibitor low molecular weight dextran sulfate prevents TLR4-induced phenotypic and functional maturation of human dendritic cells. in Journal of immunology (Baltimore, Md. : 1950) 2008
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Human Polyclonal ICAM-1 Primary Antibody for ELISA, WB - ABIN1532900
Simmons, Makgoba, Seed: ICAM, an adhesion ligand of LFA-1, is homologous to the neural cell adhesion molecule NCAM. in Nature 1988
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study reveals that ICAM-1 E469K polymorphism is not involved in the susceptibility of acromegaly but may be associated with fasting plasma glucose, systolic blood pressure, diastolic blood pressureand HDL (show HSD11B1 Antibodies)-C, the cardiovascular risk factors of acromegaly
Data suggest that urine intercellular adhesion molecule-1 (uICAM-1) is a biomarker of renal disorder, and might have the potential to predict which patients will require surgery.
Denbinobin inhibited IL-1beta (show IL1B Antibodies)-induced ICAM-1/VCAM-1 (show VCAM1 Antibodies) expression and monocyte adhesion to osteoarthritis fibroblast-like synoviocytes via mirn146-a
Pregnant women with gestational diabetes are characterized by higher concentration of s-ICAM-1, reflecting activation and dysfunction of endothelial cells.
Hypochlorite-modified albumin (show ALB Antibodies)-induced ICAM-1 expression is caused by the activation of a redox-sensitive intracellular signal cascade involving ERK1/2 (show MAPK1/3 Antibodies) and p38(MAPK (show MAPK14 Antibodies)), culminating in the activation of NF-kappaB (show NFKB1 Antibodies) and involving NOXs among the upstream signals
BD patients have a lower expression of ICAM-1, VCAM-1 (show VCAM1 Antibodies), and E-selectin (show SELE Antibodies) in their tumor tissues. BD-HDL (show HSD11B1 Antibodies) facilitates the adhesion of tumor cells to vascular endothelium by upregulating the expression of ICAM-1 and VCAM-1 (show VCAM1 Antibodies), thereby promoting the initial progression of breast cancer metastasis
ICAM-1 polymorphism rs1799969 is significantly associated with increased susceptibility to overall cancer.
Secondhand smoke exposure (measured by hair nicotine) was associated with increased soluble ICAM1 in children and adolescents.
TLR2 (show TLR2 Antibodies) S450S and ICAM1 K469E polymorphisms may be associated with polycystic ovary syndrome and metabolic comorbidities in obese women.
ICAM1 expression and release is elevated during cellular oxidative stress and is enhanced by TNF (show TNF Antibodies).
ICAM1 regulates pathological cardiac remodeling by mediating proinflammatory leukocyte infiltration in the left ventricle and cardiac fibrosis and dysfunction.
Its antiatherogenic impact might be associated with a suppressed adhesion to the endothelium due to down-regulation of endothelial ICAM-1 expression.
Luteolin may inhibit tumor angiogenesis and tumor cell proliferation by down-regulation of LFA- 3 and PCNA (show PCNA Antibodies) and up-regulation of ICAM-1 in tumor tissue of tumor-bearing mice, thereby achieving its anti-tumor effect.
our results further highlight the pleiotropic role of ICAM-1 in T-cell-dependent immune responses, with a major role in Treg cell development and suppressive function.
the present results shed light on regulation of expression and function of ICAM-1 on neutrophils and identify it as an additional regulator of neutrophil effector responses in host defense.
we identified intracellular adhesion molecule (ICAM)-1, through fibrinogen binding, as a previously unreported mediator of platelet-monocyte interactions
miR (show MLXIP Antibodies)-222 was transported into recipient endothelial cells by endothelial microparticles and functionally regulated expression of its target protein ICAM-1 in vitro and in vivo.
Timely blockade of ICAM-1.LFA-1 (show ITGAL Antibodies) interaction prevents disease onset in a mouse model of emphysema.
MRTF-A (show MKL1 Antibodies)/B depletion results in an increase in the cell surface expression of ICAM-1 and interactions between HAoECs and leukocytes
neutrophils are reliant on endothelial activation for adhesion, initially via E-selectin (show SELE Antibodies) and subsequently, by integrin-mediated binding to ICAM-1 and VCAM-1 (show VCAM1 Antibodies), combined with CXCL8 (show IL8 Antibodies)-dependent chemotaxis.
ADMA has potent adverse effects on cell proliferation, intracellular ROS (show ROS1 Antibodies) generation, cell permeability, levels of ICAM-1, and the tight-junction protein occludin (show OCLN Antibodies)
Neutrophil lung infiltrations in porcine reproductive and respiratory syndrome virus infection infected animals is both ICAM-1 dependent and independent.
Chitosan oligosaccharides downregulate the expression of E-selectin (show SELE Antibodies) and ICAM-1 by inhibiting the phosphorylation of Mitogen-Activated Protein Kinases and the activation of NF-kappaB (show NFKB1 Antibodies) in lipopolysaccharides treated porcine iliac artery endothelial cells.
Data show that all five molecules, BNP (show BNC2 Antibodies), ICAM-1, TNF-alpha (show TNF Antibodies), VCAM-1 (show VCAM1 Antibodies) and IL-6 (show IL6 Antibodies), quickly and reliably signaled adverse interactions.
Altered shear stress stimulates upregulation of endothelial VCAM-1 (show VCAM1 Antibodies) and ICAM-1 in a BMP-4 (show BMP4 Antibodies)- and TGF-beta1 (show TGFB1 Antibodies)-dependent pathway.
ICAM1 and IL10 (show IL10 Antibodies) were upregulated in ventilator-induced lung injury. Nuclear transcription factor AP-1 (show JUN Antibodies) may be responsible for this upregulation.
Hepatocellular glycogen (show GYS1 Antibodies) decreases the expression of ICAM-1 mRNA of hepatic stellate cells.
This gene encodes a cell surface glycoprotein which is typically expressed on endothelial cells and cells of the immune system. It binds to integrins of type CD11a / CD18, or CD11b / CD18 and is also exploited by Rhinovirus as a receptor.
intercellular adhesion molecule 1
, intercellular adhesion molecule 1-like
, cell surface glycoprotein P3.58
, intercellular adhesion molecule 1 (CD54), human rhinovirus receptor
, major group rhinovirus receptor
, leukocyte adhesion molecule