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The protein encoded by IL25 is a cytokine that shares sequence similarity with interleukin 17. Additionally we are shipping IL25 Antibodies (227) and IL25 Proteins (58) and many more products for this protein.
Showing 10 out of 63 products:
Human IL25 ELISA Kit for Sandwich ELISA - ABIN365233
Hams, Armstrong, Barlow, Saunders, Schwartz, Cooke, Fahy, Crotty, Hirani, Flynn, Voehringer, McKenzie, Donnelly, Fallon: IL-25 and type 2 innate lymphoid cells induce pulmonary fibrosis. in Proceedings of the National Academy of Sciences of the United States of America 2014
Inhibition of IL-25 resulted in decreased type 2 T cells and macrophages in the primary tumor microenvironments, both reported to enhance breast tumor invasion and subsequent metastasis to the lung.
this study shows that activation of primed TH2 cells occurs independently of group 2 innate lymphoid cells or their cytokines but that the effector function of both cell types was dependent on combinatorial exposure to IL-33 (show IL33 ELISA Kits), IL-25 and TSLP (show TSLP ELISA Kits)
These data demonstrate that IL-25 is critical for host protective immunity against H. polygyrus bakeri infection, highlighting its potential application as a therapeutic agent against parasitic nematode infection worldwide.
Findings indicate that the release of interleukin 25 (IL-25) from tumour-associated fibroblasts (TAFs) may serve as a check point for control of mammary tumour metastasis.
IL-25 and CD4(+) TH2 cells enhance type 2 innate lymphoid cell-derived IL-13 production, which promotes IgE-mediated experimental food allergy.
IL-22 (show IL22 ELISA Kits) restrains tapeworm-mediated protection against colitis via regulation of IL-25 expression
IL-25 promotes the function of CD4 (show CD4 ELISA Kits)+CD25 (show IL2RA ELISA Kits)+ T regulatory cells and prolongs skin-graft survival in murine models
IL-25 and IL-33 (show IL33 ELISA Kits) are critical for induction of Th2-type cytokine-mediated allergic airway eosinophilia, but not Th17-type cytokine-mediated airway neutrophilia, at the local sites of lungs in the challenge phase of mice sensitized EC with OVA
Suggest that chronic exposure of murine airways to IL-25 alone is able to reproduce a local angiogenic milieu in asthmatic mice.
we have demonstrated that the expression of ET-1 (show EDN1 ELISA Kits) and IL-25 was coordinately upregulated in the lesional keratinocytes of patients with AD and a murine AD model.
Our findings suggest that the IL-25/IL-25R axis may play an important role in promoting the recruitment and proinflammatory function of eosinophils in allergic asthma.
Chronic rhinosinusitis with nasal polyps (CRSwNP) epithelial cells release TSLP (show TSLP ELISA Kits) and IL-25 when stimulated by poly(I:C) but not by DP or AF, suggesting that viral infection may contribute to maintain and amplify the T2 immune response seen in CRSwNP.
Increased induction and expression of TSLP (show TSLP ELISA Kits), IL-25, and IL-33 (show IL33 ELISA Kits) from nasal epithelial cells contribute to the pathophysiology of eosinophilic chronic rhinosinusitis.
Women with endometriosis showed significantly higher levels of IL-25 in their PF (p=0.019) compared to controls. IL-25 levels did not correlate with the stage of endometriosis.
IL-8 (show IL8 ELISA Kits) and IL-25 are targets of miR (show MLXIP ELISA Kits)-20b.
Our findings indicate that the alarmin cytokines IL-33 (show IL33 ELISA Kits) and IL-25 increase basophil activation and migratory potential, and may pose as a novel therapeutic targets for the treatment of allergic asthma.
The IL17E rs79877597 SNP was a modifier of the risk for psoriasis disease severity and psoriatic arthritis.
Therefore, IL-17E/IL-17RB signaling represents a potential therapeutic target for treatment of hepatocellular carcinoma
The protein encoded by this gene is a cytokine that shares sequence similarity with interleukin 17. This cytokine can induce NF-kappaB activation, and stimulate the production of interleukin 8. Both this cytokine and interleukin 17B are ligands for the cytokine receptor IL17BR. Studies of a similar gene in mice suggest that this cytokine may be a pro-inflammatory cytokine favoring the Th2-type immune response. Alternative splicing results in multiple transcript variants.
, interleukin 25-like
, UPF0556 protein C19orf10 homolog
, interleukin 17E