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MAOA is one of two neighboring gene family members that encode mitochondrial enzymes which catalyze the oxidative deamination of amines, such as dopamine, norepinephrine, and serotonin. Additionally we are shipping Monoamine Oxidase A Antibodies (88) and Monoamine Oxidase A Proteins (10) and many more products for this protein.
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Results provide novel evidence that blood MAOA expression predicts alcohol consumption and that heavy alcohol use is linked to low MAOA expression in both the blood and NAc core.
study found the ability to consolidate sleep during the dark cycle was disrupted by prenatal iron deprivation, specifically in monkeys with the low-MAOA genotype
The effects of iron deficiency are dependent on MAOA genotype in terms of both direction and size of the effect.
rhMAOA-LPR (show FAS ELISA Kits) allele modifies the effects of maternal temperament on offspring's personality development.
No statistically significant differences were found between cases and controls for the allele frequencies in five genes: TH, SLC18A2 (show Slc18a2 ELISA Kits), DRD1 (show DRD1 ELISA Kits), DRD3 (show DRD3 ELISA Kits) and COMT (show COMT ELISA Kits). Conversely, some alleles of the 12 sNPs from the DRD2 (show DRD2 ELISA Kits) locus and the 5 from the MAOA locus showed significant associations with excessive alcohol consumption.
Aggressive behavior arising from childhood maltreatment is moderated by MAOA-VNTR, which may be differentially sensitive to the subtype of childhood maltreatment experienced, among Chinese adolescents
In a sample of Chinese Han adolescents, MAOA and COMT (show COMT ELISA Kits) genes both interacted with positive parenting in their associations with reactive but not proactive aggression.
discussion of genetic and environmental factors involved in the regulation of MAO-A expression, in the contexts of neuropsychiatric function and of the regulation of neuronal survival and death (review).
These findings suggest that the interaction of DRD2 (show DRD2 ELISA Kits) rs1079597 and MAOA rs309850 3-repeat affects smoking intensity in young Taiwanese men.
Polymorphisms in the monoamine oxidase A (MAOA) and catechol-O-methyltransferase (COMT (show COMT ELISA Kits)) genes modify cognitive impairment and psychiatric symptoms in Huntington's disease patients.
A functional promoter polymorphism, MAOA-LPR (show FAS ELISA Kits), plays a role in determining continuity of parent-rated attention problems during adolescence
the association of SLC6A4 (show SLC6A4 ELISA Kits) and MAOA genes with measures of obesity
evidence of a moderating effect of the MAOA gene on antisocial outcomes in a population-based sample of young males; higher risks for antisocial outcomes were observed in males carrying the MAOA low-frequency alleles in comparison with high-frequency allele carriers for most outcomes when exposed to violence
Results emphasize the importance of childhood as a sensitive period in which accumulating adversity might increase the vulnerability to externalizing psychopathology in MAOA-L males and MAOA-H females
cloning and characterization of monoamine oxidase A and B genes in pig
Two pools of ten bovine embryos, comprised of the 4-, 8- to 16-cell, morula, blastocyst, and expanded blastocyst stages, were collected. Total RNA was isolated, and the RT-PCR-RFLP technique was used to observe expression of the MAOA gene.
MAOA was seriously demethylated or showed aberrant methylation patterns in four aborted clones.
data lead us to conclude that elevation of AP-1 (show JUN ELISA Kits) or NF-kB indirectly decreases MAO-A protein levels which, in turn, diminishes MAO-A ability in the VTA of the mesolimbic dopaminergic pathway
Huntington disease (show HTT ELISA Kits) neural cells exhibit increased Monoamine oxidase-A and Monoamine oxidases-B expression and activity
acute stress induces anxiety-like responses by affecting rapid dendritic remodeling in the pyramidal cells of orbitofrontal cortex and basolateral amygdala; furthermore, our data show that MAO-A and monoamine metabolism are required for these phenomena.
These findings demonstrate that regulation of monoamine levels by Mao (show MAO ELISA Kits) activity in beta cells is pivotal for physiological insulin (show INS ELISA Kits) secretion and that loss of MaoB (show MAOB ELISA Kits) expression may contribute to the beta cell dysfunction in type 2 diabetes.
Results suggest a role for KLF11 (show KLF11 ELISA Kits) in upregulating MAO-A in depressive disorder and chronic social stress, suggesting that inhibition of the pathways regulated by this transcription factor may aid in the therapeutics of neuropsychiatric illnesses
The results of this study suggest that heightened dACC and amygdala activation and their connectivity are neuroaffective mechanisms underlying anger control in participants with the low-functioning allele of the MAOA gene.
These results suggest that early developmental enhancements in 5-HT (show DDC ELISA Kits) levels have long-term effects on the modulation of behavioral flexibility associated with MAO-A deficiency.
Under conditions of chronic hemodynamic stress, enhanced MAO-B (show MAOB ELISA Kits) activity is a major determinant of cardiac structural and functional disarrangement.
Knockdown of MAO-A expression in embryos induces high serotonin levels and abnormal brain development, which can be rescued by inactivation of serotonin receptor-6.
Both monoamine oxidase A (and B) knockout mice displayed neuropathological alterations typical of autism-spectrum disorders.
This gene is one of two neighboring gene family members that encode mitochondrial enzymes which catalyze the oxidative deamination of amines, such as dopamine, norepinephrine, and serotonin. Mutation of this gene results in Brunner syndrome. This gene has also been associated with a variety of other psychiatric disorders, including antisocial behavior. Alternatively spliced transcript variants encoding multiple isoforms have been observed.
monoamine oxidase A
, amine oxidase [flavin-containing] A-like
, amine oxidase [flavin-containing] A
, monoamine oxidase type A
, putative monoamine oxidase A