Browse our anti-CACNA1H (CACNA1H) Antibodies

Full name:
anti-Calcium Channel, Voltage-Dependent, T Type, alpha 1H Subunit Antibodies (CACNA1H)
On are 63 Calcium Channel, Voltage-Dependent, T Type, alpha 1H Subunit (CACNA1H) Antibodies from 12 different suppliers available. Additionally we are shipping CACNA1H Proteins (9) and CACNA1H Kits (4) and many more products for this protein. A total of 76 CACNA1H products are currently listed.
alpha13.2, CACNA1HB, Cav3.2, ECA6, EIG6, MNCb-1209
list all antibodies Gene Name GeneID UniProt
CACNA1H 8912 O95180
CACNA1H 58226  
CACNA1H 114862 Q9EQ60

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anti-Human CACNA1H Antibodies:

anti-Mouse (Murine) CACNA1H Antibodies:

anti-Rat (Rattus) CACNA1H Antibodies:

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Top referenced anti-CACNA1H Antibodies

  1. Human Monoclonal CACNA1H Primary Antibody for AA, ICC - ABIN361762 : Chen, Lu, Pan, Zhang, Wu, Xu, Liu, Jiang, Bao, Yao, Ding, Lo, Qiang, Chan, Shen, Wu: Association between genetic variation of CACNA1H and childhood absence epilepsy. in Annals of neurology 2003 (PubMed)
    Show all 3 references for 361762

  2. Human Monoclonal CACNA1H Primary Antibody for ICC, IHC (fro) - ABIN447362 : García-Caballero, Gadotti, Stemkowski, Weiss, Souza, Hodgkinson, Bladen, Chen, Hamid, Pizzoccaro, Deage, François, Bourinet, Zamponi: The deubiquitinating enzyme USP5 modulates neuropathic and inflammatory pain by enhancing Cav3.2 channel activity. in Neuron 2014 (PubMed)
    Show all 2 references for 447362

More Antibodies against CACNA1H Interaction Partners

Human Calcium Channel, Voltage-Dependent, T Type, alpha 1H Subunit (CACNA1H) interaction partners

  1. CACNA1H variant is associated with differential antiepileptic drug response in childhood absence epilepsy.

  2. There is a direct link between CACNA1H(M1549V) mutation and an increased aldosterone production. This suggests that calcium channel blockers may be beneficial in the treatment of a subset of patients with primary aldosteronism.

  3. CACNA1H might be a susceptibility gene predisposing to PA with different phenotypic presentations, opening new perspectives for genetic diagnosis and management of patients with PA.

  4. heterozygous mutations identified in a pediatric patient with chronic pain and absence seizures result in loss of channel function, with significantly smaller current densities across a wide range of voltages when co-expressed in tsA (show PRDX2 Antibodies)-201 cells.

  5. CaV3.1 (show CACNA1G Antibodies), CaV3.2 and CaV3.3 (show CACNA1I Antibodies) channels, are best recognized for their negative voltage of activation and inactivation thresholds that allow them to operate near the resting membrane potential of neurons.

  6. Study revealed no association between the 15 tagSNPs of CACNA1A (show CACNA1A Antibodies), 1C, and 1H and antiepileptic drug efficacy in the Chinese Han epileptic population; the TAGAA haplotype of CACNA1A (show CACNA1A Antibodies) may be a risk factor for drug resistance

  7. Cav3.2 channels are highly phosphorylated in the mammalian brain and establish phosphorylation as an important mechanism involved in the dynamic regulation of Cav3.2 channel gating properties

  8. Recurrent gain of function mutation in calcium channel CACNA1H causes early-onset hypertension with primary aldosteronism.

  9. reveal an unexpected role of CaV3.2 channels in regulating NMDA-R-mediated transmission and a novel epileptogenic mechanism for human childhood absence epilepsy

  10. The I-II loop of the Cav3.2 protein inhibits neuronal Cav3.1 (show CACNA1G Antibodies) and Cav3.2 channels.

Mouse (Murine) Calcium Channel, Voltage-Dependent, T Type, alpha 1H Subunit (CACNA1H) interaction partners

  1. The important roles of the CaV (show CA5A Antibodies) 3.2 T-type calcium channels in myogenic tone.

  2. findings show that 2 Amyotrophic lateral sclerosis (ALS)-associated missense mutations produce alterations on the channel activity, consistent with a loss of channel function; findings implicate CACNA1H as a susceptibility gene in one form of ALS

  3. findings suggest that chronic intermittent hypoxia leads to an augmented calcium influx via reactive oxygen species -dependent facilitation of CaV3.2 protein trafficking to the plasma membrane.

  4. these data show that CaV3.2 T-type channels have prev8iously unrecognized roles in supporting the meiotic-maturation-associated increase in ER Ca(2 (show CA2 Antibodies)+) stores and mediating Ca(2 (show CA2 Antibodies)+) influx required for the activation of development.

  5. MTF1 (show MTF1 Antibodies) mediates the increase of CaV3.2 mRNA and a rise in intracellular Zn(2+) which is associated with status epilepticus.

  6. both suramin and gossypetin produced dose-dependent and long-lasting mechanical anti-hyperalgesia that was abolished or greatly attenuated in Cav3.2 null mice

  7. Data show increased expression of T-type Ca(2 (show CA2 Antibodies)+) current and association of protein kinase C alpha (PKCalpha (show PKCa Antibodies)) with caveolin-3 (Cav-3 (show CAV3 Antibodies))was disrupted in the hypertrophic ventricular myocyte.

  8. the asymmetric effects of the Cav3.2 and its partial reversal by behavior training on the hippocampal transcriptome

  9. This study demonstrated that CaV3.2 KO mice have altered retinal waves but normal direction selectivity.

  10. Suggest a negative feedback mechanism of the myogenic response in which CaV3.2 channel modulates downstream ryanodine receptor (show RYR3 Antibodies)-BKCa (show KCNMA1 Antibodies) to hyperpolarize and relax arteries.

CACNA1H Antigen Profile

Antigen Summary

This gene encodes a T-type member of the alpha-1 subunit family, a protein in the voltage-dependent calcium channel complex. Calcium channels mediate the influx of calcium ions into the cell upon membrane polarization and consist of a complex of alpha-1, alpha-2/delta, beta, and gamma subunits in a 1:1:1:1 ratio. The alpha-1 subunit has 24 transmembrane segments and forms the pore through which ions pass into the cell. There are multiple isoforms of each of the proteins in the complex, either encoded by different genes or the result of alternative splicing of transcripts. Alternate transcriptional splice variants, encoding different isoforms, have been characterized for the gene described here. Studies suggest certain mutations in this gene lead to childhood absence epilepsy (CAE).

Alternative names and synonyms associated with CACNA1H

  • calcium channel, voltage-dependent, T type, alpha 1H subunit (CACNA1H) antibody
  • calcium channel, voltage-dependent, T type, alpha 1H subunit (Cacna1h) antibody
  • alpha13.2 antibody
  • CACNA1HB antibody
  • Cav3.2 antibody
  • ECA6 antibody
  • EIG6 antibody
  • MNCb-1209 antibody

Protein level used designations for CACNA1H

calcium channel, voltage-dependent, T type, alpha 1Hb subunit , low-voltage-activated calcium channel alpha1 3.2 subunit , low-voltage-activated calcium channel alpha13.2 subunit , voltage dependent t-type calcium channel alpha-1H subunit , voltage-dependent T-type calcium channel subunit alpha-1H , voltage-gated calcium channel alpha subunit Cav3.2 , voltage-gated calcium channel alpha subunit CavT.2 , voltage-gated calcium channel subunit alpha Cav3.2 , T-type Cav3.2 , calcium channel alpha13.2 subunit

8912 Homo sapiens
58226 Mus musculus
114862 Rattus norvegicus
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