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Human Complement 4 ELISA Kit for Sandwich ELISA - ABIN2683588
Sahu, Lambris: Structure and biology of complement protein C3, a connecting link between innate and acquired immunity. in Immunological reviews 2001
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Human Complement 4 ELISA Kit for Competition ELISA - ABIN2683861
Yang, Chung, Zhou, Lhotta, Hebert, Birmingham, Rovin, Yu: The intricate role of complement component C4 in human systemic lupus erythematosus. in Current directions in autoimmunity 2004
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Human Complement 4 ELISA Kit for Sandwich ELISA - ABIN365889
Hou, Qi, Liao, Zhang, Fang, Zhou, Liu, Bai, Zhang, Kijlstra, Yang: Copy number variations of complement component C4 are associated with Behçet's disease but not with ankylosing spondylitis associated with acute anterior uveitis. in Arthritis and rheumatism 2013
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Human Complement 4 ELISA Kit for Sandwich ELISA - ABIN612679
Koehler, Swain, Sanderson, Krishnan, Watt, Charlton: Growth hormone, dehydroepiandrosterone and adiponectin levels in non-alcoholic steatohepatitis: an endocrine signature for advanced fibrosis in obese patients. in Liver international : official journal of the International Association for the Study of the Liver 2012
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Human Complement 4 ELISA Kit for Sandwich ELISA - ABIN414468
Ma, Cui, Liao, Zhao: Complement activation contributes to the injury and outcome of kidney in human anti-glomerular basement membrane disease. in Journal of clinical immunology 2013
Human Complement 4 ELISA Kit for Competition ELISA - ABIN612676
Roos, Mollenhauer, Démant, Rittner: A molecular basis for the two locus model of human complement component C4. in Nature 1982
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The C4A (show C4B ELISA Kits)-complete transcript is the main transcript under normal physiological conditions, while C4A (show C4B ELISA Kits)-AS is augmented when mastitis develops.
An elevated number of C4 genes was observed in Alzheimer's disease (AD) patients as compared with healthy controls. The presence of high C4A (show C4B ELISA Kits) and C4B (show C4B ELISA Kits) copy numbers in AD patients could explain the increased C4 protein expression observed in AD patients, thus highlighting a possible role for C4A (show C4B ELISA Kits) and C4B (show C4B ELISA Kits) copy number variations in the risk of developing AD.
The structural basis for inhibition of the classical and lectin complement pathways by S. aureus extracellular adherence protein binding to C4b (show C4B ELISA Kits) has been presented.
The aims of this study were to elucidate the copy number variations of C4A (show C4B ELISA Kits) and C4B (show C4B ELISA Kits) in relation to disease risk in systemic lupus erythematosus, and to compare the basis of race-specific C4A (show C4B ELISA Kits) deficiency between East Asians and individuals of European descent
C4A (show C4B ELISA Kits) and C4B (show C4B ELISA Kits) gene copy numbers are stronger risk factors for juvenile-onset than for adult-onset systemic lupus erythematosus
Data indicate Ig-like transcript 4 (ILT4 (show LILRB2 ELISA Kits)) as a cellular receptor for complement split products (CSPs) complement component 4d (C4d).
Suggest a prominent prognostic value of C4d staining as a rejection marker in ABO compatible kidney transplantation.
The interaction between C4BP (show C4BPA ELISA Kits) and Leptospira interrogans LcpA, LigA and LigB is sensitive to ionic strength and inhibited by heparin.
Schizophrenia risk from complex variation of complement component 4 (show C4A ELISA Kits)
There was a significant association between C4d deposition on allograft endothelial cells and presence of class II DSA I chronic liver allograft failure.
Carrying no copies of C4B (show C4B ELISA Kits) significantly increases the risk of cCSC, whereas carrying three C4B (show C4B ELISA Kits) copies is protective.
This gene encodes the basic form of complement factor 4, part of the classical activation pathway. The protein is expressed as a single chain precursor which is proteolytically cleaved into a trimer of alpha, beta, and gamma chains prior to secretion. The trimer provides a surface for interaction between the antigen-antibody complex and other complement components. The alpha chain may be cleaved to release C4 anaphylatoxin, a mediator of local inflammation. Deficiency of this protein is associated with systemic lupus erythematosus. This gene localizes to the major histocompatibility complex (MHC) class III region on chromosome 6. Varying haplotypes of this gene cluster exist, such that individuals may have 1, 2, or 3 copies of this gene. In addition, this gene exists as a long form and a short form due to the presence or absence of a 6.4 kb endogenous HERV-K retrovirus in intron 9.
MHC-linked complement C4
, complement C4
, complement component 4 (within H-2S)
, complement component 4, gene 1
, complement component 4B (Chido blood group)
, complement component 4B (Childo blood group)
, C3 and PZP-like alpha-2-macroglobulin domain-containing protein 3
, Chido form of C4
, basic complement C4
, complement C4-B
, complement C4B1a