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anti-Human FZD3 Antibodies:
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Human Monoclonal FZD3 Primary Antibody for CyTOF, FACS - ABIN4898967
Baksh, Boland, Tuan: Cross-talk between Wnt signaling pathways in human mesenchymal stem cells leads to functional antagonism during osteogenic differentiation. in Journal of cellular biochemistry 2007
Show all 3 Pubmed References
Human Monoclonal FZD3 Primary Antibody for CyTOF, FACS - ABIN4898966
Wong, He, Chan, Chan, Wong, Cheung, Luk, Au, Chiu, Ma, Chan: Clinical significance of frizzled homolog 3 protein in colorectal cancer patients. in PLoS ONE 2013
Show all 3 Pubmed References
Human Polyclonal FZD3 Primary Antibody for ELISA - ABIN408707
You, Nguyen, Albers, Lin, Holcombe: Wnt pathway-related gene expression in inflammatory bowel disease. in Digestive diseases and sciences 2008
Human Polyclonal FZD3 Primary Antibody for IF (p), IHC (p) - ABIN1714171
Su, Zhang, Gan, Jiang, Xiao, Zou, Li, Jiang: Deregulation of the planar cell polarity genes CELSR3 and FZD3 in Hirschsprung disease. in Experimental and molecular pathology 2016
Human Monoclonal FZD3 Primary Antibody for ELISA, WB - ABIN521713
Wang, Tekpetey, Kidder: Identification of WNT/beta-CATENIN signaling pathway components in human cumulus cells. in Molecular human reproduction 2009
marked reduction in the prominence of TUJ1 bundles in number, thickness, and length. Our results showed that deregulation of the planar cell polarity genes CELSR3 (show CELSR3 Antibodies) and FZD3 might disrupt the enteric innervation pattern
seven-transmembrane domain receptors Celsr3 (show CELSR3 Antibodies) and Fzd3, in particular, control the development of most longitudinal tracts in the central nervous system. [Review]
Our analysis showed no significant association between the rs2241802 polymorphism in FZD3 gene and neural tube defects
DNA methylation (show HELLS Antibodies) aberrations rather than polymorphisms of FZD3 gene increase the risk of spina bifida in a high-risk region for neural tube defects.
Wnt3a (show WNT3A Antibodies)/Frizzled-3 signaling plays important role in regulating the proliferation and differentiation of neural crest cells and various developmental stages of melanocyte precursors.
Aberrant methylation modification of the FZD3 gene increases the risk of congenital hydrocephalus (show FOXC1 Antibodies) by altering chromatin structure and disturbing gene expression.
FZD3 signaling sensitized peripheral sensory neurons in pain hypersensitivity.
clinical significance of frizzled homolog 3 protein in colorectal cancer patients
Genetic variants of the FZD3 gene may affect susceptibility to schizophrenia in Chinese Han and Va populations.
This study found a significant association between schizophrenia and the FZD3 gene in single nucleotide polymorphisms and haplotype analyses.
loss of Fzd3 induces severe malformations of the developing eye and this defect is phenocopied by loss of the activated leukocyte cell adhesion molecule (Alcam (show ALCAM Antibodies)).
Frizzled3 is required to shape the pattern of RBC (show CACNA1C Antibodies) somas and dendrites, and the structural and functional connectivity between rods and RBCs. Our results highlight novel functions for Fzd3 in regulating retinal development.
Temporal and spatial expression profiles of Frizzled 3 in the ovary during the estrous cycle has been reported.
Celsr3 (show CELSR3 Antibodies) and Fzd3 enable immature neurons to respond to Wnt7 (show WNT7B Antibodies), upregulate Jag1 (show JAG1 Antibodies) and thereby facilitate feedback signals that tune the timing of neural progenitor cell fate decisions via Notch (show NOTCH1 Antibodies) activation.
This study demonstrated that Frizzled3 Controls Axonal Polarity and Intermediate Target Entry during Striatal Pathway Development.
In Fz3(-/-) limbs, dorsal axons stall at a precise location in the nerve plexus, and, in contrast to the phenotypes of several other axon path-finding mutants, Fz3(-/-) dorsal axons do not reroute to other trajectories
Fz3 and Fz6 (show FZD6 Antibodies) have partly interchangeable roles in tissue polarity signaling for epithelial orientation and axon growth and guidance
The first signal, controlled by cadherin, EGF (show EGF Antibodies)-like, laminin G-like, seven-pass, G-type receptor (Celsr) 2 (show CELSR2 Antibodies), Celsr3 (show CELSR3 Antibodies), Frizzled3 (Fzd3) and Van Gogh (show VANGL2 Antibodies) like2 (Vangl2 (show VANGL2 Antibodies)) organizes multicilia in individual cells (single-cell polarity)
Celsr2 (show CELSR2 Antibodies)-3 and Fzd3 regulate axonal navigation in the forebrain by using mechanisms different from classical epithelial core planar cell polarity, and require interacting partners other than Vangl1 (show Vangl1 Antibodies)-2 that remain to be identified.
Several central nervous system axon tracts require Fz3 function as early as embryonic day 11.5, and that Fz3 is required for pathfinding by dopaminergic and serotonergic axons in the brain and by a subset of optic tract axons.
This gene is a member of the frizzled gene family. Members of this family encode seven-transmembrane domain proteins that are receptors for the wingless type MMTV integration site family of signaling proteins. Most frizzled receptors are coupled to the beta-catenin canonical signaling pathway. The function of this protein is unknown, although it may play a role in mammalian hair follicle development. Alternative splicing results in multiple transcript variants. This gene is a susceptibility locus for schizophrenia.
frizzled 3, seven transmembrane spanning receptor
, frizzled homolog 3
, frizzled homolog 3 (Drosophila)
, frizzled 3
, frizzled family receptor 3