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Putative adapter protein involved in asymmetrical cell division and cell polarization processes.
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artitioning defective 3-like protein may serve as a promising prognostic marker and a potential therapeutic target for colorectal cancer treatment. Further study is necessary to understand the regulatory mechanism of partitioning defective 3-like protein in colorectal cancer and the feasibility of its application in clinic.
Par3L interacts with Lkb1 (show STK11 Proteins) and regulates the activity of AMPK (show PRKAA1 Proteins) signaling cascade.
The association of single-nucleotide polymorphisms of PARD3B with slower progression of HIV infection to AIDS is reported.
PAR3beta, expressed intrinsically or extrinsically, localizes to the tight junctions, indicating that the localization does not require the ternary complex formation.
binding of 14-3-3 (show YWHAQ Proteins) to Par3beta is dependent on phosphorylation
Par3L is expressed by multipotent stem cells in the terminal end buds of murine mammary glands. Ablation of Par3L resulted in rapid and profound stem cell loss.
Atp6ap2/(P)RR (show ATP6AP2 Proteins) interacted with partitioning defective 3 homolog (PAR3 (show F2RL2 Proteins)) protein, which is known to function in the Par (show AFG3L2 Proteins)-atypical protein kinase C (show PRKCZ Proteins)
Putative adapter protein involved in asymmetrical cell division and cell polarization processes. May play a role in the formation of epithelial tight junctions.
, amyotrophic lateral sclerosis 2 (juvenile) chromosome region, candidate 19
, amyotrophic lateral sclerosis 2 chromosomal region candidate gene 19 protein
, par-3 partitioning defective 3 homolog B
, partitioning defective 3 homolog B
, partitioning defective 3-like protein
, partitioning-defective 3-beta
, amyotrophic lateral sclerosis 2 chromosomal region candidate gene 19 protein homolog