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Bcl-2 antibody (AA 5-205) (HRP)

This anti-Bcl-2 antibody is a Rabbit Polyclonal antibody detecting Bcl-2 in ELISA. Suitable for Mouse.
Catalog No. ABIN7176967

Quick Overview for Bcl-2 antibody (AA 5-205) (HRP) (ABIN7176967)

Target

See all Bcl-2 (BCL2) Antibodies
Bcl-2 (BCL2) (B-Cell CLL/lymphoma 2 (BCL2))

Reactivity

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  • 37
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  • 1
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Mouse

Host

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Rabbit

Clonality

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Polyclonal

Conjugate

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This Bcl-2 antibody is conjugated to HRP

Application

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ELISA
  • Binding Specificity

    • 55
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    • 11
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    AA 5-205

    Cross-Reactivity

    Mouse

    Purification

    >95%, Protein G purified

    Immunogen

    Recombinant Mouse Apoptosis regulator Bcl-2 protein (5-205AA)

    Isotype

    IgG
  • Application Notes

    Optimal working dilution should be determined by the investigator.

    Restrictions

    For Research Use only
  • Format

    Liquid

    Buffer

    Preservative: 0.03 % Proclin 300
    Constituents: 50 % Glycerol, 0.01M PBS, PH 7.4

    Preservative

    ProClin

    Precaution of Use

    This product contains ProClin: a POISONOUS AND HAZARDOUS SUBSTANCE which should be handled by trained staff only.

    Storage

    -20 °C,-80 °C

    Storage Comment

    Upon receipt, store at -20°C or -80°C. Avoid repeated freeze.
  • Target

    Bcl-2 (BCL2) (B-Cell CLL/lymphoma 2 (BCL2))

    Alternative Name

    Bcl2

    Background

    Background: Suppresses apoptosis in a variety of cell systems including factor-dependent lymphohematopoietic and neural cells. Regulates cell death by controlling the mitochondrial membrane permeability. Appears to function in a feedback loop system with caspases. Inhibits caspase activity either by preventing the release of cytochrome c from the mitochondria and/or by binding to the apoptosis-activating factor (APAF-1).

    Aliases: Bcl2 antibody, Bcl-2 antibody, Apoptosis regulator Bcl-2 antibody

    UniProt

    P10417

    Pathways

    MAPK Signaling, PI3K-Akt Signaling, Apoptosis, Caspase Cascade in Apoptosis, Regulation of Muscle Cell Differentiation, Cell-Cell Junction Organization, Skeletal Muscle Fiber Development, Autophagy, Smooth Muscle Cell Migration, Negative Regulation of intrinsic apoptotic Signaling
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