Recombinant AGT antibody (Alexa Fluor 488)

Catalog No. ABIN7792560

The Rabbit Monoclonal anti-AGT antibody is suitable to detect AGT in samples from Human. It has been validated for WB, IP, ICC, IF and FACS.

Quick Overview for Recombinant AGT antibody (Alexa Fluor 488) (ABIN7792560)

Target: AGT (Angiotensinogen (serpin Peptidase Inhibitor, Clade A, Member 8) (AGT))

Antibody Type: Recombinant Antibody

Reactivity: Human

Host: Rabbit

Clonality: Monoclonal

Conjugate: This AGT antibody is conjugated to Alexa Fluor 488

Application: Western Blotting (WB), Immunoprecipitation (IP), Immunocytochemistry (ICC), Immunofluorescence (IF), Flow Cytometry (FACS)

Product Details anti-AGT Antibody (Alexa Fluor 488)

Purpose: Conjugated Angiotensinogen Rabbit mAb

Specificity: Angiotensinogen Antibody detects endogenous levels of total Angiotensinogen

Purification: Affinity-chromatography

Immunogen: A synthesized peptide derived from human Angiotensinogen

Application Details

Application Notes: most applications: 1: 50 - 1: 250

Restrictions: For Research Use only

Handling

Buffer: Liquid in PBS, 0.5 % BSA, 40 % Glycerol, 0.02 % Sodium Azide.

Preservative: Sodium azide

Precaution of Use: This product contains Sodium azide: a POISONOUS AND HAZARDOUS SUBSTANCE which should be handled by trained staff only.

Handling Advice: Protect from light!

Storage: 4 °C

Storage Comment: Store at 4°C in dark for 6 months

Target Details for AGT

Target: AGT (Angiotensinogen (serpin Peptidase Inhibitor, Clade A, Member 8) (AGT))

Alternative Name: Angiotensinogen

Background: AGT, Ang, Ang I, Ang II, Ang III, Angiotensinogen, ANHU, ANRT, AT-2, AT-II, PAT, Pre angiotensinogen, Serpin A8, SERPINA8,

UniProt: P01019

Pathways: JAK-STAT Signaling, ACE Inhibitor Pathway, EGFR Signaling Pathway, Peptide Hormone Metabolism, Regulation of Systemic Arterial Blood Pressure by Hormones, Regulation of Lipid Metabolism by PPARalpha, Protein targeting to Nucleus, Feeding Behaviour, Monocarboxylic Acid Catabolic Process, Dicarboxylic Acid Transport, Positive Regulation of Response to DNA Damage Stimulus, Regulation of long-term Neuronal Synaptic Plasticity