Potassium Inwardly-Rectifying Channel, Subfamily J, Member 12 (KCNJ12) (Middle Region) Peptide
KCNJ12
Reactivity: Human
Host: Synthetic
BP, WB
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- Target See all Kir2.2 (KCNJ12) products
- Kir2.2 (KCNJ12) (Potassium Inwardly-Rectifying Channel, Subfamily J, Member 12 (KCNJ12))
- Protein Region
- Middle Region
- Origin
- Human
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Source
- Synthetic
- Application
- Blocking Peptide (BP), Western Blotting (WB)
- Characteristics
- This is a synthetic peptide designed for use in combination with anti-KCNJ12 antibody (Catalog #: ARP35315_P050). It may block above mentioned antibody from binding to its target protein in western blot and/or immunohistochecmistry under proper experimental settings. There is no guarantee for its use in other applications.
- Purification
- Purified
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- Application Notes
- Each Investigator should determine their own optimal working dilution for specific applications.
- Restrictions
- For Research Use only
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- Format
- Lyophilized
- Reconstitution
- Add 100 μL of sterile PBS. Final peptide concentration is 1 mg/mL in PBS.
- Concentration
- 1 mg/mL
- Buffer
- Final peptide concentration is 1 mg/mL in PBS.
- Handling Advice
- Avoid repeated freeze-thaw cycles.
- Storage
- -20 °C
- Storage Comment
- For longer periods of storage, store at -20°C. Avoid repeat freeze-thaw cycles.
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- Target
- Kir2.2 (KCNJ12) (Potassium Inwardly-Rectifying Channel, Subfamily J, Member 12 (KCNJ12))
- Synonyms
- IRK-2 Peptide, IRK2 Peptide, KCNJN1 Peptide, Kir2.2 Peptide, Kir2.2v Peptide, hIRK Peptide, hIRK1 Peptide, hkir2.2x Peptide, kcnj12x Peptide, Kir2.1 Peptide, IRK Peptide, KIR2.2 Peptide, MB-IRK2 Peptide, potassium voltage-gated channel subfamily J member 12 Peptide, ATP-sensitive inward rectifier potassium channel 12 Peptide, potassium inwardly-rectifying channel, subfamily J, member 12 Peptide, KCNJ12 Peptide, LOC746628 Peptide, Kcnj12 Peptide
- Background
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KCNJ12 is an inwardly rectifying K+ channel which may be blocked by divalent cations. This protein is thought to be one of multiple inwardly rectifying channels which contribute to the cardiac inward rectifier current (IK1). This gene is located within the Smith-Magenis syndrome region on chromosome 17. This gene encodes an inwardly rectifying K+ channel which may be blocked by divalent cations. This protein is thought to be one of multiple inwardly rectifying channels which contribute to the cardiac inward rectifier current (IK1). The gene is located within the Smith-Magenis syndrome region on chromosome 17. Publication Note: This RefSeq record includes a subset of the publications that are available for this gene. Please see the Entrez Gene record to access additional publications. PRIMARYREFSEQ_SPAN PRIMARY_IDENTIFIER PRIMARY_SPAN COMP 1-486 BC027982.1 12-497 487-488 AC068418.5 101651-101652 489-2455 BC027982.1 500-2466 2456-2483 DA115102.1 132-159 2484-2485 AC068418.5 141899-141900 2486-2889 DA115102.1 161-564 2890-3454 BM799671.1 118-682 3455-3457 AC068418.5 142870-142872 3458-4692 AK024229.1 926-2160 4693-5230 AK024229.1 2163-2700
Alias Symbols: FLJ14167, IRK2, KCNJN1, Kir2.2, Kir2.2v, hIRK, hIRK1, hkir2.2x, kcnj12x, IRK-2
Protein Interaction Partner: ABLIM1,APBA1,CASK,DLG1,DLG2,DLG3,DLG4,DMD,DTNA,LIN7A,LIN7B,LIN7C,PRKACA,SNTA1,SNTB1,SNTB2,ABLIM1,APBA1,CASK,DLG1,DLG2,DLG3,DLG4,DMD,DTNA,LIN7A,LIN7B,LIN7C,MPP6,SNTA1,SNTB1,SNTB2
Protein Size: 433 - Molecular Weight
- 49 kDa
- Gene ID
- 3768
- NCBI Accession
- NM_021012, NP_066292
- UniProt
- Q14500
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