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Nonvoltage-gated, amiloride-sensitive, sodium channels control fluid and electrolyte transport across epithelia in many organs. Additionally we are shipping SCNN1B Proteins (7) and SCNN1B Kits (4) and many more products for this protein.
Showing 10 out of 164 products:
Hamster Polyclonal SCNN1B Primary Antibody for ICC, IF - ABIN2486410
Elias, Rafii, Rahman, Otulakowski, Cutz, OBrodovich: The role of alpha-, beta-, and gamma-ENaC subunits in distal lung epithelial fluid absorption induced by pulmonary edema fluid. in American journal of physiology. Lung cellular and molecular physiology 2007
Show all 6 references for ABIN2486410
Hamster Polyclonal SCNN1B Primary Antibody for ICC, IF - ABIN2486415
Kakizoe, Kitamura, Ko, Wakida, Maekawa, Miyoshi, Shiraishi, Adachi, Zhang, Masilamani, Tomita: Aberrant ENaC activation in Dahl salt-sensitive rats. in Journal of hypertension 2009
Show all 6 references for ABIN2486415
Hamster Polyclonal SCNN1B Primary Antibody for ICC, IF - ABIN2486416
Gu: Effect of [Cl(-)]i on ENaC activity from mouse cortical collecting duct cells. in Journal of cellular physiology 2008
Show all 6 references for ABIN2486416
Hamster Polyclonal SCNN1B Primary Antibody for ICC, IF - ABIN2486409
Bruns, Hu, Ahn, Sheng, Hughey, Kleyman: Multiple epithelial Na+ channel domains participate in subunit assembly. in American journal of physiology. Renal physiology 2003
Show all 6 references for ABIN2486409
Hamster Polyclonal SCNN1B Primary Antibody for ICC, IF - ABIN2486408
Mauro, Guitard, Behne, Oda, Crumrine, Komuves, Rassner, Elias, Hummler: The ENaC channel is required for normal epidermal differentiation. in The Journal of investigative dermatology 2002
Show all 6 references for ABIN2486408
Hamster Polyclonal SCNN1B Primary Antibody for ICC, IF - ABIN2486411
Dyka, May, Enz: Subunits of the epithelial sodium channel family are differentially expressed in the retina of mice with ocular hypertension. in Journal of neurochemistry 2005
Show all 6 references for ABIN2486411
Human Polyclonal SCNN1B Primary Antibody for WB - ABIN1881778
Song, Wei, Zhou, Lazrak, Liu, Londino, Squadrito, Matalon: Inhibition of lung fluid clearance and epithelial Na+ channels by chlorine, hypochlorous acid, and chloramines. in The Journal of biological chemistry 2010
Show all 3 references for ABIN1881778
Human Polyclonal SCNN1B Primary Antibody for IF (p), IHC (p) - ABIN713471
Pasham, Pathare, Fajol, Rexhepaj, Michael, Pakladok, Alesutan, Rotte, Föller, Lang: OSR1-sensitive small intestinal Na+ transport. in American journal of physiology. Gastrointestinal and liver physiology 2012
Cow (Bovine) Polyclonal SCNN1B Primary Antibody for WB - ABIN2776090
Snyder, Olson, Kabra, Zhou, Steines: cAMP and serum and glucocorticoid-inducible kinase (SGK) regulate the epithelial Na(+) channel through convergent phosphorylation of Nedd4-2. in The Journal of biological chemistry 2004
These results indicated a significant association between EH and SCNN1B methylation, which was affected by age, gender and antihypertensive therapy.
Three nonsynonymous amino acid variants in SCNN1B in nonwhite Cystic fibrosis (show S100A8 Antibodies) patients with non-diagnostic CFTR (show CFTR Antibodies) genotypes was
These results do not suggest an important role for the T594M variant of the ENaC (show SCNN1A Antibodies) gene contributing to either the development or severity of hypertension in subjects of Indo (show IDO1 Antibodies)-Aryan ancestry.
The transcriptional expression of alpha, beta, and gamma subunits of ENaC (show SCNN1A Antibodies) was elevated in nasal polyp compared to nasal mucosa
causative mutation for Liddle's syndrome (LS) in this patient was identified to be a novel frameshift mutation. DNA sequencing resulted in exon 13 of SCNN1B gene: SCNN1B NM_000336.2:c.1 724_1730dupGGCCCAC [p.Pro5 75Argfs*17].
analysis of a novel frameshift mutation in the betaENaC gene in an isolated case of Liddle syndrome
Suggest that SCNN1B gene has important roles in blood pressure regulation in the Han Chinese population.
significant association between the rs3743966 SNP in intron 12 of epithelial sodium channel beta-subunit and essential hypertension
Suggest that beta-ENaC mediates cytotrophoblast migration and increasing beta-ENaC expression by HO-1 (show HMOX1 Antibodies) induction enhances migration.
ENaCbeta but not ENaCalpha (show SCNN1A Antibodies) expression is significantly lower in severe preeclampsia compared with normal pregnancy.
Nedd4-2 (show NEDD4L Antibodies)-mediated ubiquitination of beta-ENaC leading to endocytosis and degradation of apical Na(+) channels is a key feature of hypoxia-induced inhibition of transepithelial alveolar Na(+) transport.
Data indicate that insulin receptor (InsR (show INSR Antibodies)) is important for epithelial sodium channel (ENaC (show SCNN1A Antibodies)) activity.
Our findings are consistent with the role of betaENaC as a vascular smooth muscle cell mechanosensor and function of evolutionarily related nematode degenerin (show ACCN1 Antibodies) proteins.
These data suggest that adenylyl cyclase VI mediates vasopressin (show AVP Antibodies)-stimulated ENaC (show SCNN1A Antibodies) activity in the kidney.
Cys (show DNAJC5 Antibodies) substitutions at sites in the wrist domains of the beta and gamma subunits alter the Na+ self-inhibition response.
ENaC (show SCNN1A Antibodies) is not a passive passenger in regulated epithelial vesicle trafficking, but plays a role in establishing and maintaining the pool of vesicles that respond to cAMP stimulation.
These findings demonstrate that myogenic constriction in afferent arterioles is dependent on normal expression of betaENaC
loss of beta-ENaC associated with early signs of renal injury and increased mean arterial pressure
generation of transgenic mice with airway-specific overexpression of the epithelial Na(+) channel (ENaC (show SCNN1A Antibodies)) is an alternative approach to mimic cystic fibrosis (show S100A8 Antibodies) ion transport pathophysiology in the lung
Airway surface liquid volume regulation determines different airway phenotypes in liddle compared with betaENaC-overexpressing mice.
Nonvoltage-gated, amiloride-sensitive, sodium channels control fluid and electrolyte transport across epithelia in many organs. These channels are heteromeric complexes consisting of 3 subunits: alpha, beta, and gamma. This gene encodes the beta subunit, and mutations in this gene have been associated with pseudohypoaldosteronism type 1 (PHA1), and Liddle syndrome.
sodium channel, nonvoltage-gated 1, beta
, sodium channel, nonvoltage-gated 1, beta (Liddle syndrome)
, amiloride-sensitive sodium channel subunit beta
, amiloride-sensitive sodium channel subunit beta 1
, epithelial Na(+) channel subunit beta
, epithelial sodium channel beta-2 subunit
, epithelial sodium channel beta-3 subunit
, nasal epithelial sodium channel beta subunit
, nonvoltage-gated sodium channel 1 subunit beta
, Amiloride-sensitive sodium channel subunit beta
, epithelial amiloride-sensitive sodium channel beta subunit
, epithelial sodium channel ENaC beta subunit
, epithelial sodium channel, beta subunit
, amiloride sensitive epithelial sodium ion channel beta subunit
, epithelium sodium channel beta subunit
, ENaC beta
, amiloride sensitive sodium channel beta1 subunit
, sodium channel, nonvoltage-gated, type I, beta