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mouse homolog is an extracellular matrix protein; human homolog is associated with hereditary corneal dystrophies. Additionally we are shipping Transforming Growth Factor, beta Induced Antibodies (16) and Transforming Growth Factor, beta Induced Proteins (12) and many more products for this protein.
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This study shows that upregulation of bigh3 and bambia mRNA correlated with the process of fin regeneration and regulation of TGF-b signaling, suggesting a new role for these proteins.
Results describe the distribution of TGFBI (show TGFB1 ELISA Kits) in the developing skeletal muscle of zebrafish embryos, and use antisense morpholino oligonucleotides to investigate the function of the endogenous protein.
TGF-beta (show TGFB1 ELISA Kits)-induced matricellular protein-null lungs displayed diminished elastic recoil and gas exchange efficiency
the data suggest a model by which cytokine-induced Myt3 suppression leads to Tgfbi de-repression and subsequently to impaired islet-cell migration, revealing a novel role for Myt3 in regulating islet-cell migration.
Studies reveal that growth hormone (GH (show GH1 ELISA Kits))increases TGFBI (show TGFB1 ELISA Kits) expression in a mouse and rat models of diabetic nephropathy (DN) which correlate with clinical results, and suggest that this mechanism can explain the podocytes depletion in DN.
Tgfbi (show TGFB1 ELISA Kits) deficiency is associated with the degradation of cartilage matrix
The results support a novel inflammatory pathway for diabetic retinopathy. This pathway is initiated by TGFbeta (show TGFB1 ELISA Kits) released from macrophages, which promotes synthesis and release of BIGH3 (show TGFB1 ELISA Kits) protein by REC (show ZDHHC2 ELISA Kits) and REC (show ZDHHC2 ELISA Kits) apoptosis.
TGFBI (show TGFB1 ELISA Kits) is a potential risk gene for human diabetes.
High blood TGFBIp levels were observed in cecal ligation and puncture (CLP)-induced septic mice. The absence of the TGFBIp gene in mice attenuated CLP-induced sepsis.
Collectively, our findings indicate that betaig-h3 (show TGFB1 ELISA Kits) promotes the fibrogenic influence of TGFbeta (show TGFB1 ELISA Kits) signaling, neutralizing the prochondrogenic influence of the hypoxic-inducible factor 1 activated by the microenvironment of limb mesenchymal aggregates.
the observed effect of TGFBI (show TGFB1 ELISA Kits) on bone mass and bone size was largely caused by the effect of TGFBI (show TGFB1 ELISA Kits) on periosteal bone formation
betaig-h3 (show TGFB1 ELISA Kits) bears alphavbeta3 integrin-interacting motifs that mediate endothelial cell adhesion and migration and angiogenesis
mouse homolog is an extracellular matrix protein\; human homolog is associated with hereditary corneal dystrophies
, transforming growth factor-beta-induced protein ig-h3
, transforming growth factor, beta induced, 68 kDa
, beta ig-h3
, transforming growth factor, beta induced