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LIPC encodes hepatic triglyceride lipase, which is expressed in liver. Additionally we are shipping Lipase, Hepatic Kits (55) and Lipase, Hepatic Proteins (15) and many more products for this protein.
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The expression of hHL promoted hepatic triglyceride accumulation and de novo lipogenesis without affecting triglyceride secretion, and this was associated with an upregulation of Srebf1 (show SREBF1 Antibodies) as well as the main genes controlling the synthesis of fatty acids. Transgenic mice also exhibited more adiposity and an increased LPL (show LCP1 Antibodies)-mediated FFA influx into the WAT without affecting glucose tolerance
LPL (show LCP1 Antibodies) is important for the maturation of small discoidal HDL (show HSD11B1 Antibodies) particles into large spherical HDL (show HSD11B1 Antibodies) particles, while HL is important for HDL (show HSD11B1 Antibodies) remodeling of very large HDL (show HSD11B1 Antibodies) particles into intermediate-size HDL (show HSD11B1 Antibodies) particles, as shown in lipoprotein lipase (show LPL Antibodies) and hepatic lipase deficiency
The expression level of LIPC, SLC16A8 (show MCT3 Antibodies), and TIMP-3 (show TIMP3 Antibodies) was significantly associated with age-related macular degeneration pathology.
We found inverse independent correlations between metabolic clearance rate of insulin (show INS Antibodies) (MCRI) and hepatic lipase (P = 0.0004), insulin (show INS Antibodies) secretion (P = 0.0002), alanine aminotransferase (show ALT Antibodies) (P = 0.0045), total fat mass (P = 0.014), and diabetes (P = 0.03). MCRI and apolipoprotein A-I (show APOA1 Antibodies) exhibited a positive independent correlation (P = 0.035).
Hepatic lipase activity increases after levothyroxine therapy in subclincal hypothyroidism and can be interpreted as a possible explanation for the decrease in remnant-like lipoprotein cholesterol.
The hepatic lipase (LIPC) rs10468017 variant was associated with a significantly decreased risk of AMD (show AMD1 Antibodies)
the association between LIPC C-514T polymorphism, obesity and plasma lipid profile in Chinese children and adolescents, is reported.
results show that SM is a physiological inhibitor of HL activity in lipoproteins and that the specificity of the enzyme towards HDL (show HSD11B1 Antibodies) is at least partly due to its low SM content.
The role of hepatic triglyceride lipase (show LIPG Antibodies) in remnant lipoprotein metabolism.
Dietary fat intake significantly modified the LIPC genetic effect on changes in serum triglyceride, LDL cholesterol, and HDL (show HSD11B1 Antibodies) cholesterol concentrations during a 2-y randomized weight-loss intervention trial.
The LIGHT (Tumour necrosis factor (show TNF Antibodies) ligand superfamily member 14, TNFSF14 (show TNFSF14 Antibodies))/Lymphotoxin beta-Receptor (show LTBR Antibodies)(LTbeta (show LTB Antibodies)-R) pathway, which is involved in T-cell and macrophage activation, was diminished in plasma and in apoE (show APOE Antibodies)-/-Irs2 (show IRS2 Antibodies)+/-HL-/- atheromas.
hepatic lipase KO mice had dyslipidemia including hypercholesterolemia, hypertriglyceridemia and increased non-esterified fatty acid levels. Also glucose intolerance, pancreatic and hepatic inflammation and steatosis.
Bone marrow (BM)-derived hepatic lipase mitigates the HDL (show HSD11B1 Antibodies)-lowering, HDL (show HSD11B1 Antibodies)-modulating, and cholesterol-raising effects of BM-derived cholesteryl ester transfer protein (show CETP Antibodies).
HL expressed by osteoblasts has an impact on osteoblast OPG (show TNFSF11 Antibodies) expression and that lack of HL leads to increased bone mass.
Hepatic lipase and endothelial lipase (show LIPG Antibodies) influence molecular species of several classes of plasma lipids.
interaction of LIGHT with LTbetaR on hepatocytes, but not Kupffer cells, is sufficient to down regulate hepatic lipase expression and that this effect can be independent of LIGHT's costimulatory function.
Hepatic lipase- and endothelial lipase (show LIPG Antibodies)-deficiency in mice promotes macrophage-to-feces RCT (show FOXE3 Antibodies) and HDL (show HSD11B1 Antibodies) antioxidant properties.
Data show a novel pathway involving HL hydrolysis of VLDL that activates PPARdelta (show PPARD Antibodies) through generation of specific monounsaturated fatty acids.
These results demonstrated that fatty acid synthase (show FASN Antibodies) and hepatic lipase might be FXR (show NR1H4 Antibodies)-regulated genes in liver cells.
The additive effect of hepatic lipase and endothelial lipase (show LIPG Antibodies) on HDL (show HSD11B1 Antibodies) metabolism but not macrophage reverse cholesterol transport in mice.
LIPC encodes hepatic triglyceride lipase, which is expressed in liver. LIPC has the dual functions of triglyceride hydrolase and ligand/bridging factor for receptor-mediated lipoprotein uptake.
hepatic triacylglycerol lipase
, hepatic triglyceride lipase
, lipase, hepatic
, lipase C
, hepatic lipase
, hepatic triacylglycerol lipase-like
, lipase member C