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Human Polyclonal ADRB1 Primary Antibody for ELISA, WB - ABIN185584
Chandra, Portbury, Ray, Ream, Groelle, Chikaraishi: Beta1-adrenergic receptors maintain fetal heart rate and survival. in Biology of the neonate 2006
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Human Polyclonal ADRB1 Primary Antibody for IF (p), IHC (p) - ABIN669351
Deng, Liu, Zhang, Wang, Peng, Wei, Jiang: Exogenous norepinephrine attenuates the efficacy of sunitinib in a mouse cancer model. in Journal of experimental & clinical cancer research : CR 2014
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Dog (Canine) Polyclonal ADRB1 Primary Antibody for WB - ABIN2774823
Tsai, Lee, Chen, Kao, Lu, Lin, Chen, Chen: Testosterone replacement increases aged pulmonary vein and left atrium arrhythmogenesis with enhanced adrenergic activity. in International journal of cardiology 2014
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Human Polyclonal ADRB1 Primary Antibody for ELISA, WB - ABIN269984
Wallukat, Muñoz Saravia, Haberland, Bartel, Araujo, Valda, Duchen, Diaz Ramirez, Borges, Schimke: Distinct patterns of autoantibodies against G-protein-coupled receptors in Chagas' cardiomyopathy and megacolon. Their potential impact for early risk assessment in asymptomatic Chagas' patients. in Journal of the American College of Cardiology 2010
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Human Polyclonal ADRB1 Primary Antibody for IF (p) - ABIN669355
Feridooni, Hotchkiss, Baguma-Nibasheka, Zhang, Allen, Chinni, Pasumarthi: Effects of β-adrenergic receptor drugs on embryonic ventricular cell proliferation and differentiation and their impact on donor cell transplantation. in American journal of physiology. Heart and circulatory physiology 2017
Report associations between ADRB1 SNPs and acute coronary syndrome/cardiovascular risk factors in Arab population.
The downregulation of beta-ARs (show SLURP1 Antibodies) after ureter dilation, particularly for beta1-AR and beta3-AR in the muscular layer, suggests a potential compensatory mechanism involving increased contraction of the ureter to push urine through the obstruction.
The results demonstrate that Gly/Gly polymorphism in Arg389Gly ADRB1 was an independent risk factor for hypertension; moreover, the patients who carried the Gly389Gly genotype had a significantly improved metoprolol antihypertensive effect than those with ADRB1.
All patients carrying the Val158-COMT (show COMT Antibodies) allele exhibited higher preoperative norepinephrine concentrations. Moreover, we found that both beta1-adrenoceptor polymorphisms were associated with a longer stay in hospital. We conclude that patients with the Val158-COMT (show COMT Antibodies) genotype exhibit higher endogenous resting plasma norepinephrine levels.
two barcodes are involved in sorting Wild Type ss1-AR out of early endosomes.
Data suggest that the partial agonist STD (show SULT2A1 Antibodies)-101-D1 of beta-1 adrenergic receptor (ADRB1) is an research tool to study mechanisms of G protein-coupled receptor (show ADRA1A Antibodies) signal transduction.
Analyzing the functional relevance of individual sites using phosphosite-deficient receptor mutants we found phosphorylation of the ADRB1 at Ser461/Ser462 in the distal part of the C-terminus to determine beta-arrestin2 (show ARRB2 Antibodies) recruitment and receptor internalization
Studied frequency of ADRB1 Arginine389Glycine (Arg389Gly) and Cytochrome p450 2D6 (show CYP2D6 Antibodies)*10 (Cyp2D6 (show CYP2D6 Antibodies)*10) polymorphism in cases of heart failure-reduced ejection fraction (HFREF), and to evaluate the influence of the polymorphisms in response to beta-blocker (BB) therapy; found a statistically significant association was observed with CC genotype and Glisin-Glisin (GG) genotype.
Among individuals with previous small artery ischemic stroke, the ADRB1 Gly49 polymorphism was associated with major adverse cardiovascular events, particularly small artery ischemic stroke, a risk that may be increased among beta-blocker-treated individuals.
ADRB1 SNPs were associated with myocardial infarction susceptibility, blood pressure and plasma lipids.
Zebrafish larvae lacking beta1AR expression by morpholino knockdown displayed lower heart rates than control fish, whereas larvae deficient in both beta2aAR and beta2bAR expression exhibited significantly higher heart rates than controls.
Cardiac specific expression of the mutated Cav1.2PKA_P2(-/-) gene reduces Cav1.2 alpha1c protein concentration, calcium current (ICa), and the beta-adrenergic stimulation of L-type ICa in cardiomyocytes.
the present study demonstrated that cardiac contractility/relaxation and heart rate is increased in beta1AR TG and beta2AR (show ADRB2 Antibodies) TG mice, and indicated that this increase may be related to the overexpression of G proteins and Gproteinassociated proteins.
ghrelin (show GHRL Antibodies) has a critical role in preventing hypoglycemia and promoting survival during severe caloric restriction, a process that requires cell-expressed beta1AR
knocking out of the beta1/2 (show TFAP2B Antibodies) receptor significantly diminished the ST25 acupuncture-induced inhibition of gastric motility and jejunal motility without significantly altering the enhancement of colonic motility induced by acupuncture at ST25.
Our findings that BAG3 (show BAG3 Antibodies) is localized at the sarcolemma and t-tubules while modulating myocyte contraction and action potential duration through specific interaction with the beta1-adrenergic receptor and L-type Ca(2 (show CA2 Antibodies)+) channel provide novel insight into the role of BAG3 (show BAG3 Antibodies) in cardiomyopathies and increased arrhythmia risks in heart failure.
Galphai2 (show GNAI2 Antibodies) deficiency combined with cardiac beta1-adrenoceptor overexpression strongly impaired survival and cardiac function, leading to dilated cardiomyopathy.
Knockout of the C5aR1 (show C5AR1 Antibodies) attenuated the effect of beta1-AR in the heart, suggesting an association between the beta1-AR and C5aR1 (show C5AR1 Antibodies), although further investigation is required to determine if this is a direct or causal association
Partial gap junction uncoupling increased propensity and amplitude of Ca(2 (show CA2 Antibodies)+) alternans, and made them more sensitive to reversal by beta-AR activation, as in isolated myocytes.
These data reveal a novel interplay between the E2F pathway, beta2-adrenergic/PKA/PDE4D, and ERK/c-Src axis in fine tuning the pathological hypertrophic growth response.
Myocardial adrenergic receptor beta 1 preferentially associates with AC5 (show ADCY5 Antibodies).
The adrenergic receptors (subtypes alpha 1, alpha 2, beta 1, and beta 2) are a prototypic family of guanine nucleotide binding regulatory protein-coupled receptors that mediate the physiological effects of the hormone epinephrine and the neurotransmitter norepinephrine. Specific polymorphisms in this gene have been shown to affect the resting heart rate and can be involved in heart failure.
adrenergic, beta-1-, receptor
, beta-1 adrenergic receptor
, beta-1 adrenoceptor
, beta-1 adrenoreceptor
, adrenergic receptor, beta 1
, beta 1-adrenergic receptor beta 1-AR
, beta 1 adrenergic receptor
, beta 1 adrenergic protein
, Beta-1 adrenergic receptor
, G-protein-coupled hormone receptor
, beta 1-adrenergic receptor
, beta-1-adrenergic receptor
, tripartite motif-containing 75
, beta1-adrenergic G-protein-coupled receptor
, beta 1-AR
, cardiac beta adrenergic receptor