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Human Polyclonal SLC25A4 Primary Antibody for ELISA - ABIN559869
Loers, Makhina, Bork, Dörner, Schachner, Kleene et al.: The interaction between cell adhesion molecule L1, matrix metalloproteinase 14, and adenine nucleotide translocator at the plasma membrane regulates L1-mediated neurite outgrowth of murine cerebellar ... in The Journal of neuroscience : the official journal of the Society for Neuroscience 2012
These findings suggest that DRAK1 translocates in response to stimuli and induces apoptosis through its interaction with specific binding partners, p53 and/or ANT2.
Yeast aac2 (show SLC25A5 Antibodies) R96H and aac2 (show SLC25A5 Antibodies) R252G mutations are equivalent to R80H and R235G human ANT1 pathological mutations. mtDNA instability induced by aac2R96H and aac2R252G is rescued by N-acetylcysteine.
identification by whole-exome sequencing of seven probands harboring dominant, de novo SLC25A4 mutations; all affected individuals presented at birth, were ventilator dependent and, where tested, revealed severe combined mitochondrial respiratory chain deficiencies associated with a marked loss of mitochondrial DNA copy number in skeletal muscle
A directed proteomic approach discovered the novel interaction of BKCa (show KCNMA1 Antibodies) with Tom22 (show TOMM22 Antibodies), a component of the mitochondrion outer membrane import system, and the adenine nucleotide translocator (ANT).
Identification of ZNF555 (show ZNF555 Antibodies) as a putative transcriptional factor that impacts ANT1 promoter activity in facioscapulohumeral dystrophy myoblasts.
elevated ANT1 expression supports EV infection and is associated with EV persistence, a condition with adverse prognosis.
Data suggest acetylation of ANT1 at lysines 10/23/92 has dramatic physiological effects on ADP-ATP exchange; extent of acetylation of lysine 23 decreases following physical activity; this change is highly dependent on insulin (show INS Antibodies) sensitivity/resistance.
Expression of ANT1 were lower in inclusion body myositis samples versus both polymyositis and controls
Compares and contrasts all the known human SLC25A (show SLC25A25 Antibodies)* genes and includes functional information.
A 13-generation Mennonite pedigree with autosomal recessive myopathy and cardiomyopathy due to an SLC25A4 frameshift null mutation (c.523delC, p.Q175RfsX38), which codes for the heart-muscle isoform of the adenine nucleotide translocator-1, was studied.
ANT1 can induce autoimmune myocarditis in A/J mice by generating autoreactive T cells.
miR (show MLXIP Antibodies)-2861 and ANT1 are regulators of cardiomyocyte necrosis and myocardial infarction.
These results suggest that follicular helper T cells cells and IL-21 (show IL21 Antibodies) might be involved in the pathogenesis of viral myocarditis and play an important role in anti-ANT autoantibody production.
Data indicate that palmitoyl-carnitine (PC) increases the type 2 ryanodine receptor (RyR2 (show RYR2 Antibodies)) oxidation and reduces the ATP/ADP translocase (ANT) activity.
Downregulation of adenine nucleotide translocator 1 exacerbates tumor necrosis factor-alpha (show TNF Antibodies)-mediated cardiac inflammatory responses.
Plasma membrane-localized ANT1 and ANT2 (show SLC25A5 Antibodies) regulate L1-mediated neurite outgrowth in conjunction with MMP14 (show MMP14 Antibodies).
Although the molecular mechanism linking ANT1-Cys (show DNAJC5 Antibodies)(57) nitroalkylation and uncoupling is not yet known, these data suggest that ANT1-mediated uncoupling may be a mechanism for nitroalkene-induced cardioprotection.
The attenuation of ANT-1 in the presence of PGC-1alpha overexpression preserves the mitochondrial membrane potential in response to hydrogen-peroxide stress.
Data demonstrate that ANT1 protein levels are upregulated in Mecp2-null mice.
uncoupling protein-3 (show UCP3 Antibodies) can minimize the induction of the adenine nucleotide translocase (show SLC25A5 Antibodies)-mediated 'energy-wasting' process during Calorie restriction (CR).
Data show that the binding sites on adenine nucleotide translocator 1 (ANT1) have an affinity for cardiolipin (CL).
This gene is a member of the mitochondrial carrier subfamily of solute carrier protein genes. The product of this gene functions as a gated pore that translocates ADP from the mitochondrial matrix into the cytoplasm. The protein forms a homodimer embedded in the inner mitochondria membrane. Mutations in this gene have been shown to result in autosomal dominant progressive external opthalmoplegia and familial hypertrophic cardiomyopathy.
ADP,ATP carrier protein 1
, ADP,ATP carrier protein, heart/skeletal muscle
, ADP/ATP translocase 1
, adenine nucleotide translocator 1 (skeletal muscle)
, heart/skeletal muscle ATP/ADP translocator
, solute carrier family 25 member 4
, ANT 1
, adenine nucleotide translocase-1
, adenine nucleotide translocator 1, skeletal muscle
, adenine nucleotide translocator 1
, mitochondrial adenine nucleotide translocator
, solute carrier family 25 (mitochondrial adenine nucleotide translocator) member 4
, ADP,ATP carrier protein, heart
, ADP/ATP carrier
, ADP/ATP translocase T1
, ADP,ATP carrier protein 1, mitochondrial
, 30 kDa CSQ-binding protein
, 30 kDa calsequestrin-binding protein
, CSQ-binding 30 kDa protein
, solute carrier family 25 (mitochondrial carrier; adenine nucleotide translocator), member 5
, solute carrier family 25 (mitochondrial carrier, adenine nucleotide translocator), member 4 L homeolog
, solute carrier family 25 (mitochondrial carrier; adenine nucleotide translocator), member 4 L homeolog
, solute carrier family 25 (mitochondrial carrier; adenine nucleotide translocator), member 4