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Dog (Canine) Monoclonal TLR9 Primary Antibody for CyTOF, ELISA - ABIN4360430
Gantner, Hermann, Nakashima, Matsukawa, Sakai, Bacon: CD40-dependent and -independent activation of human tonsil B cells by CpG oligodeoxynucleotides. in European journal of immunology 2003
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Dog (Canine) Monoclonal TLR9 Primary Antibody for FACS - ABIN4360435
Wang, Nicholas, Conway, Sen, Diz, Tisch, Clarke: EBV latent membrane protein 2A induces autoreactive B cell activation and TLR hypersensitivity. in Journal of immunology (Baltimore, Md. : 1950) 2006
Show all 57 Pubmed References
Dog (Canine) Monoclonal TLR9 Primary Antibody for FACS, ICC - ABIN4360434
Dillmann, Ringel, Ringleb, Mora, Olesch, Fink, Roberts, Brüne, Weigert: S1PR4 Signaling Attenuates ILT 7 Internalization To Limit IFN-α Production by Human Plasmacytoid Dendritic Cells. in Journal of immunology (Baltimore, Md. : 1950) 2016
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Dog (Canine) Monoclonal TLR9 Primary Antibody for CyTOF, ELISA - ABIN4360432
Damiano, Caputo, Bianco, DArmiento, Leonardi, De Placido, Bianco, Agrawal, Ciardiello, Tortora: Novel toll-like receptor 9 agonist induces epidermal growth factor receptor (EGFR) inhibition and synergistic antitumor activity with EGFR inhibitors. in Clinical cancer research : an official journal of the American Association for Cancer Research 2006
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Dog (Canine) Monoclonal TLR9 Primary Antibody for ELISA - ABIN4360433
Abel, Wang, Fritts, Sanchez, Chung, Fitzgerald-Bocarsly, Krieg, Miller et al.: Deoxycytidyl-deoxyguanosine oligonucleotide classes A, B, and C induce distinct cytokine gene expression patterns in rhesus monkey peripheral blood mononuclear cells and distinct alpha interferon ... in Clinical and diagnostic laboratory immunology 2005
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Human Monoclonal TLR9 Primary Antibody for FACS, IF - ABIN2191962
Rumio, Besusso, Palazzo, Selleri, Sfondrini, Dubini, Ménard, Balsari: Degranulation of paneth cells via toll-like receptor 9. in The American journal of pathology 2004
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Human Monoclonal TLR9 Primary Antibody for FACS, IF - ABIN2191963
Ahmad-Nejad, Häcker, Rutz, Bauer, Vabulas, Wagner: Bacterial CpG-DNA and lipopolysaccharides activate Toll-like receptors at distinct cellular compartments. in European journal of immunology 2002
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Human Monoclonal TLR9 Primary Antibody for FACS, IF - ABIN2191964
Rutz, Metzger, Gellert, Luppa, Lipford, Wagner, Bauer: Toll-like receptor 9 binds single-stranded CpG-DNA in a sequence- and pH-dependent manner. in European journal of immunology 2004
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Human Monoclonal TLR9 Primary Antibody for FACS - ABIN4360413
Leifer, Kennedy, Mazzoni, Lee, Kruhlak, Segal: TLR9 is localized in the endoplasmic reticulum prior to stimulation. in Journal of immunology (Baltimore, Md. : 1950) 2004
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Mouse (Murine) Polyclonal TLR9 Primary Antibody for IHC - ABIN967190
Hemmi, Takeuchi, Kawai, Kaisho, Sato, Sanjo, Matsumoto, Hoshino, Wagner, Takeda, Akira: A Toll-like receptor recognizes bacterial DNA. in Nature 2000
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Studied associations of 12 single nucleotide polymorphisms (SNPs) within toll (show TLR4 Antibodies) like receptor genes (TLR2, TLR3 (show TLR3 Antibodies), TLR4 (show TLR4 Antibodies), and TLR9) and genetic predisposition to newborn severe hepatitis.
TLR7 (show TLR7 Antibodies), TLR9, and JAK2 (show JAK2 Antibodies) genes are potential biomarkers for systemic sclerosis. High TLR7 (show TLR7 Antibodies) expression positively correlated with the late form of disease. Decreased levels of TLR9 and JAK2 (show JAK2 Antibodies) mRNA were found in the patient's cohort in comparison to non-SSc (show CYP11A1 Antibodies) individuals.
TLR9 rs187084 gene polymorphism may be associated with post-bronchiolitis wheezing, and TLR10 (show TLR10 Antibodies) rs4129009 gene polymorphism may be associated with atopy.
Meta-analysis did not show any association between TLR9 polymorphisms (rs187084, rs352140, rs5743836 and rs352139) and systemic lupus erythematosus under any model.[meta-analysis]
TLR9 contributes to tumor regression by inducing cytotoxic T cell response, reducing the numbers of myeloid-derived suppressor cells, the tumor-associated macrophages, and the regulatory T cells. It can however, also promote tumor progression and invasiveness of cervical tissue. Review.
this study shows that increased expression of TLR9 is associated with reduced DNA methylation (show HELLS Antibodies) in spontaneous preterm labor
A rapid neutralization of anticoagulant activity of NU172 was also demonstrated in fresh human whole blood 5 min after addition of AD. Furthermore, the TLR9-mediated activation of PMDC05 cells was interrupted after the addition of the R10-60 AD
BAD-LAMP (LAMP5)-silencing enhances TLR9 retention in endolysosome compartment and consequent downstream signalling events.
results suggest that TLR9 rs5743836 polymorphism is an independent risk factor for venous thromboembolism recurrence in female patients but not in males.
Based on the results, we hypothesize that aberrant surface expression of TLR9 on tumor cells may promote tumor growth and invasion. It might also highlight a dual contradictory role for CpG-ODNs, as adjutant in cancer therapy.
Our findings suggested the collaboration of TLR2-TLR9 at least in the regulation of SARM (show SARM1 Antibodies) expression.
High cholesterol intake exacerbated acetaminophen-induced acute liver injury via the accumulation of free cholesterol in the endolysosomes of liver sinusoidal endothelial cells. This accumulation enhanced Toll-like receptor 9 signaling via impairment of its membrane trafficking mechanism.
these data provide a previously uncharacterized in vivo mechanism contingent on oligodeoxy-nucleotide -administered dose, where TLR9 governs the primary response and RAGE (show AGER Antibodies) plays a distinct and cooperative function in providing a pivotal role in balancing the immune response.
this study shows that CD205 (show LY75 Antibodies)-TLR9-IL-12 (show IL12A Antibodies) axis contributes to CpG-induced oversensitive liver injury in Hepatitis B viral antigens transgenic mice by promoting the interaction of NKT (show CTSL1 Antibodies) cells with Kupffer cells
The findings support a salutary role of TLR9 in some subsets of HF conditions and underline the importance for future studies on the mechanisms of TLR9 in diastolic HF.
TLR9-driven inflammation induced a Ccr2-independent expansion of functionally enhanced extramedullary myeloid progenitors that correlated with the peripheral accumulation of monocytes in both wild-type and Ccr2(-/-) mice.
TLR9 expression on B1a cells is not critical for their IgM-dependent atheroprotection.
This study establishes a correlation between TLR-3 (show TLR3 Antibodies) and TLR-9 expression with the development of EAE. In addition, evidence of a role for the myelin peptide in targeting the innate inflammatory response to the CNS is presented.
these data demonstrate that TLR7 (show TLR7 Antibodies)/TLR9 ligands push the macrophage into a phagocytic long-lived cell, with a decreased capacity of antigen presentation and reminiscent of the M2 polarized state.
TLR9/MyD88 (show MYD88 Antibodies) signaling selectively in CD11c (show ITGAX Antibodies)(+) dendritic cells (DCs) strongly enhances murine cytomegalovirus clearance.
Porcine parvovirus virus infection significantly induced IL-6 (show IL6 Antibodies) expression and this induction depended on NF-kappaB (show NFKB1 Antibodies) activation and TLR9 signaling pathways in PK-15 cell.
TLR9 immunoreactivity is mainly detected in epithelial cells and antigen presenting cells, namely dendritic and macrophage-like cells, within the range of tissues examined. The pattern of TLR9 expression was similar in pigs of 3 weeks and 3 months of age.
These data demonstrated that TLR2, TLR3 (show TLR3 Antibodies) and TLR9 contribute to NF-kappaB (show NFKB1 Antibodies) activation in response to porcine epidemic diarrhea virus infection, but not RIG-I (show DDX58 Antibodies).
expression of TLR3 (show TLR3 Antibodies), TLR7 (show TLR7 Antibodies) and TLR9 in alveolar macrophages infected with different genotype 1 PRRSV strains
we studied TLR9 expression in lung extracts from pigs, dogs, and cattle.
Moreover, the TLR9 transfectant demonstrated its usefulness for evaluation of immunostimulation by bacterial DNA through the detection of T(H)-1, T(H)-2 type cytokine induction via TLR9 signaling.
variants in the TLR1 (show TLR1 Antibodies) gene are associated with susceptibility to bovine tuberculosis, whereas no significant association can be inferred from the polymorphisms in the TLR9 gene
mRNA abundances of TLR9, TLR2, and TLR4 together with those of beta-defensin 5 (BNBD5), an early bactericidal effector molecule of the innate system, in healthy and infected mammary glands
substantial conservation of TLR9 structure and TLR9 function in blood leukocytes
The protein encoded by this gene is a member of the Toll-like receptor (TLR) family which plays a fundamental role in pathogen recognition and activation of innate immunity. TLRs are highly conserved from Drosophila to humans and share structural and functional similarities. They recognize pathogen-associated molecular patterns (PAMPs) that are expressed on infectious agents, and mediate the production of cytokines necessary for the development of effective immunity. The various TLRs exhibit different patterns of expression. This gene is preferentially expressed in immune cell rich tissues, such as spleen, lymph node, bone marrow and peripheral blood leukocytes. Studies in mice and human indicate that this receptor mediates cellular response to unmethylated CpG dinucleotides in bacterial DNA to mount an innate immune response.
Toll-like receptor 9 protein