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anti-Human CAMK2G Antibodies:
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Human Monoclonal CAMK2G Primary Antibody for ICC, IF - ABIN361699
Barria, Muller, Derkach, Griffith, Soderling: Regulatory phosphorylation of AMPA-type glutamate receptors by CaM-KII during long-term potentiation. in Science (New York, N.Y.) 1997
Show all 14 Pubmed References
Human Polyclonal CAMK2G Primary Antibody for IHC - ABIN965741
Pak, Huang, Li, Balschun, Reymann, Chiang, Westphal, Huang: Involvement of neurogranin in the modulation of calcium/calmodulin-dependent protein kinase II, synaptic plasticity, and spatial learning: a study with knockout mice. in Proceedings of the National Academy of Sciences of the United States of America 2000
Cow (Bovine) Monoclonal CAMK2G Primary Antibody for ICC, IF - ABIN361644
Bennett, Kennedy: Deduced primary structure of the beta subunit of brain type II Ca2+/calmodulin-dependent protein kinase determined by molecular cloning. in Proceedings of the National Academy of Sciences of the United States of America 1987
Show all 13 Pubmed References
Human Polyclonal CAMK2G Primary Antibody for IF, WB - ABIN362984
Bossuyt, Helmstadter, Wu, Clements-Jewery, Haworth, Avkiran, Martin, Pogwizd, Bers: Ca2+/calmodulin-dependent protein kinase IIdelta and protein kinase D overexpression reinforce the histone deacetylase 5 redistribution in heart failure. in Circulation research 2008
Human Polyclonal CAMK2G Primary Antibody for IHC (p), WB - ABIN391313
Tombes, Krystal: Identification of novel human tumor cell-specific CaMK-II variants. in Biochimica et biophysica acta 1997
Show all 2 Pubmed References
The data suggest T287 autophosphorylation regulates substrate gating, an intrinsic property of the catalytic domain, which is amplified within the multivalent architecture of the calcium/calmodulin-dependent protein kinase II (show CAMK2 Antibodies) holoenzyme.
These findings indicate that the CaMKII-mediated GluA1 (show GRIA1 Antibodies) phosphorylation of S567 and S831 is critical for P2X2 (show P2RX2 Antibodies)-mediated AMPAR internalization and ATP-driven synaptic depression.
Laminin is instructive and CaMKII is non-permissive for the formation of complex aggregates of acetylcholine receptors on myotubes in culture.
A new molecular mechanism mediated by CAMK2gamma in intestinal epithelial cells during colitis-associated cancer (CAC (show CA2 Antibodies)) development, thereby providing a potential new therapeutic target for CAC (show CA2 Antibodies).
oxidative stress activated the TRPM2 (show CLU Antibodies)-CaMKII cascade to further induce intracellular ROS (show ROS1 Antibodies) production, which led to mitochondria fragmentation and loss of mitochondrial membrane potential
CKIalpha (show CSNK1A1 Antibodies)-mediated NS5A S235 phosphorylation is critical for HCV replication. CaMKII gamma and delta may have negative roles in the HCV life cycle.
Demonstrate that calcium/CaMKIIgamma/AKT (show AKT1 Antibodies) signaling can regulate apoptosis and autophagy simultaneously in colorectal cancer cells.
Dysfunction in CaMKII-based signaling has been linked with a host of cardiovascular phenotypes including heart failure and arrhythmia, and CaMKII levels are elevated in human and animal disease models of heart disease.
Inhibition of CaMKII activity results in an upregulation of CaMKIV (show CAMK4 Antibodies) mRNA and protein in leukemia cell lines.
CAMKIIgamma, HSP70 (show HSP70 Antibodies) and HSP90 (show HSP90 Antibodies) transcripts are differentially expressed in chronic myeloid leukemia (show BCL11A Antibodies) cells from patients with resistant mutated disease.
Redox-sensitive activation of p38 MAPK (show MAPK14 Antibodies)/HSP27 (show HSPB1 Antibodies) pathway or ERK1/2 in endothelial cells requires CaMKII
Data, including data from studies using transgenic mice, suggest that signaling via prostaglandin-F2alpha/PTGFR (show PTGFR Antibodies) and Camk2g/p38 (show CRK Antibodies)/Foxo1 (show FOXO1 Antibodies) MAP kinase (show MAPK1 Antibodies)/calcium pathways are involved in regulation of hepatic gluconeogenesis in both obesity and fasting. (Ptgfr (show PTGFR Antibodies) = prostaglandin F2alpha receptor (show PTGFR Antibodies); Camk2g = calcium-calmodulin-dependent protein kinase (show CAMK2 Antibodies) type 2 gamma; p38 (show CRK Antibodies) = p38 MAP kinase (show MAPK14 Antibodies); Foxo1 (show FOXO1 Antibodies) = forkhead box transcription factor (show FOXP2 Antibodies) O1)
RGS6 (show RGS6 Antibodies) and oxidized CaMKIIdelta together function as novel critical upstream modulators of Notch (show NOTCH1 Antibodies) signaling required for normal cardiovascular development and embryo survival.
CaMKIIgamma-deficient macrophages and atherosclerotic lesions lacking myeloid CaMKIIgamma had increased expression of the transcription factor ATF6 (show ATF6 Antibodies).
A new molecular mechanism mediated by CAMK2gamma in intestinal epithelial cells during colitis-associated cancer (CAC (show SLC25A20 Antibodies)) development, thereby providing a potential new therapeutic target for CAC (show SLC25A20 Antibodies).
Data indicate that combined cardiomyocyte-specific deletion of CaM Kinase II CaMKIIdelta/gamma does not affect ischemia/reperfusion (I/R) injury.
Cardiac CaM Kinase II genes delta and gamma contribute to adverse remodeling but redundantly inhibit calcineurin-induced myocardial hypertrophy.
CaMKIIdeltagamma couples noncanonical Wnt signaling to histone deacetylase 4 and myosin enhancer factor 2.
Camk2g-deficient obese mice have higher nuclear ATF6 (show ATF6 Antibodies) levels, and silencing ATF6 (show ATF6 Antibodies) in these mice lowers p58IPK (show DNAJC3 Antibodies) and suppresses insulin (show INS Antibodies)-induced p-Akt (show AKT1 Antibodies).
Phosphorylation at specific PEVK/titin (show TTN Antibodies) N2B-unique sequence sites was decreased in CAMK2g/d double knockout mice.
The CamkIIgamma isoform is poorly expressed in synaptic sites. Its activity is critical for receptor recycling and may provide a mechanism by which the postsynaptic acetylcholine receptor (show CHRNB1 Antibodies) density is maintained at the neuromuscular junction in vivo.
The product of this gene is one of the four subunits of an enzyme which belongs to the serine/threonine protein kinase family, and to the Ca(2+)/calmodulin-dependent protein kinase subfamily. Calcium signaling is crucial for several aspects of plasticity at glutamatergic synapses. In mammalian cells the enzyme is composed of four different chains: alpha, beta, gamma, and delta. The product of this gene is a gamma chain. Many alternatively spliced transcripts encoding different isoforms have been described but the full-length nature of all the variants has not been determined.
caMK-II subunit gamma
, calcium/calmodulin-dependent protein kinase (CaM kinase) II gamma
, calcium/calmodulin-dependent protein kinase type II subunit gamma
, caM kinase II subunit gamma
, caM-kinase II gamma chain
, calcium/calmodulin-dependent protein kinase type II gamma chain
, calcium/calmodulin-dependent protein kinase II gamma
, Ca2+/calmodulin-dependent protein kinase II
, CaMK II
, CaM kinase II gamma B
, CaM kinase II gamma C-1
, CaM kinase II gamma C-2
, CaM kinase II gamma G-1
, CaM kinase II gamma G-2
, CaM kinase II gamma J