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Glutamate receptors are the predominant excitatory neurotransmitter receptors in the mammalian brain and are activated in a variety of normal neurophysiologic processes.
Results indicate that disrupting GluA2 phosphorylation and increasing GluA2-mediated transmission in the nucleus accumbens leads to increased vulnerability to cocaine relapse.
Chronic stress-elicited depressive behavior may be due to hypertrophy of basolateral amygdala (BLA (show LACTB Antibodies)) neuronal dendrites and increased of Glur1 (show GRIA1 Antibodies)-Glur2 ratio in BLA (show LACTB Antibodies) neurons.
found the protein levels of AMPA (show GRIA3 Antibodies) receptor subunits (GluR1 (show GRIA1 Antibodies) and GluR2) are upregulated in the amygdala and the 5-HT3 receptor (show HTR3A Antibodies) is downregulated in hypothalamic regions of Socially Isolated mice.
These results provide direct evidence for cortical AMPA (show GRIA3 Antibodies) receptors to contribute to zymosan-induced visceral and spontaneous pain.
Animals trained in the trace fear conditioning protocol had GluA2 RNA editing levels were nearly 100% in amygdala and hippocampus.
These results provide evidence for VPS35 (show vps35 Antibodies)'s function in promoting spine maturation, which is likely through increasing AMPA (show GRIA3 Antibodies) receptor targeting to the postsynaptic membrane.
Bacopa monnieri extract (CDRI-08) upregulates the expression of the GluR2 subunit in the CA3 (show CA3 Antibodies) area of the hippocampus.
subcellular redistribution of GRIP1 (show NCOA2 Antibodies) and a change in the binding of GRIP1 (show NCOA2 Antibodies) to GluA2 during synaptic scaling, was observed.
Data indicate that the AMPA (show GRIA3 Antibodies) receptor subunits abundance is hippocampus, GluA2 > GluA1 (show GRIA1 Antibodies) > GluA3 (show GRIA3 Antibodies) >> GluA4 (show GRIA4 Antibodies); cortex, GluA2 > GluA3 (show GRIA3 Antibodies) >/= GluA1 (show GRIA1 Antibodies) >> GluA4 (show GRIA4 Antibodies); and cerebellum, GluA2 > GluA3 (show GRIA3 Antibodies) >/= GluA1 (show GRIA1 Antibodies) > GluA4 (show GRIA4 Antibodies).
Loss of long-term potentiation was linked to reduced N-methyl-D-aspartate (NMDA) to alpha-amino-3-hydroxy- 5-methyl-4-isoxazolepropionic acid (AMPA (show GRIA3 Antibodies)) current ratio.
Glutamate receptors are the predominant excitatory neurotransmitter receptors in the mammalian brain and are activated in a variety of normal neurophysiologic processes. This gene product belongs to a family of glutamate receptors that are sensitive to alpha-amino-3-hydroxy-5-methyl-4-isoxazole propionate (AMPA), and function as ligand-activated cation channels. These channels are assembled from 4 related subunits, Gria1-4. The subunit encoded by this gene (Gria2) is subject to RNA editing (CAG->CGG\; Q->R) within the second transmembrane domain, which is thought to render the channel impermeable to Ca(2+). Alternative splicing, resulting in transcript variants encoding different isoforms, (including the flip and flop isoforms that vary in their signal transduction properties), has been noted for this gene.
AMPA-selective glutamate receptor 2
, glutamate receptor 2
, glutamate receptor B
, AMPA glutamate receptor 2
, AMPA receptor GluR2/B
, AMPA selective glutamate receptor
, glutamate receptor ionotropic, AMPA 2