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Cytotoxicity-activating receptor that may contribute to the increased efficiency of activated natural killer (NK) cells to mediate tumor cell lysis.. Additionally we are shipping Natural Cytotoxicity Triggering Receptor 2 Antibodies (128) and Natural Cytotoxicity Triggering Receptor 2 Kits (4) and many more products for this protein.
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On CD56 (show NCAM1 Proteins)(+) CD3 (show CD3 Proteins)(-) cells, NKp44 and NKp46 (show NCR1 Proteins) expressions were high in the acute hepatitis E patients, whereas NKp30 (show NCR3 Proteins), NKp44, NKp46 (show NCR1 Proteins) and NKG2D (show KLRK1 Proteins) were high in the recovered individuals.
demonstrated that the mitogen and iK562 exposure to peripheral blood mononuclear cells can significantly improve NK activity which is co-related to the higher expression of NKp44 and NKG2D (show KLRK1 Proteins)
NCR(+) ILC3 from skin and blood of psoriasis patients produced IL-22 (show IL22 Proteins), which is regarded as a key driver of epidermal thickening, suggesting that NCR(+) ILC3 may participate in psoriasis pathology.
Balance between activating NKG2D (show KLRK1 Proteins), DNAM-1 (show CD226 Proteins), NKp44 and NKp46 (show NCR1 Proteins) and inhibitory CD94 (show KLRD1 Proteins)/NKG2A (show KLRC1 Proteins) receptors determine natural killer degranulation towards rheumatoid arthritis synovial fibroblasts.
NKp44+ ILC3 are expressed in human skin and blood and may have a role in psoriasis pathogenesis
Expression of NKp44 ligand by normal articular chondrocytes is not involved in their killing by unstimulated NK cells; however, it is responsible for anti-chondrocyte cytotoxicity mediated by long-term activated NK cells.
MLL5 is a cellular ligand for the natural cytotoxicity receptor NKp44.
Natural killer cells in HIV controller patients express an activated effector phenotype and do not up-regulate NKp44 on IL-2 (show IL2 Proteins) stimulation.
Novel interaction between proliferating cell nuclear antigen (show PCNA Proteins) and HLA I on the surface of tumor cells inhibits NK cell function through NKp44.
While interaction of TLR2 with mycobacterial cell wall promotes activation of resting NK cells and IFN-gamma (show IFNG Proteins) production, NKp44 interaction with its putative ligands could play a secondary role in maintaining cell activation
Cytotoxicity-activating receptor that may contribute to the increased efficiency of activated natural killer (NK) cells to mediate tumor cell lysis.
natural cytotoxicity triggering receptor 2
, NK cell activating receptor (NKp44)
, NK cell-activating receptor
, lymphocyte antigen 95 (activating NK-receptor; NK-p44)
, lymphocyte antigen 95 homolog (activating NK-receptor; NK-p44)
, natural killer cell p44-related protein