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Terminates the action of GABA by its high affinity sodium-dependent reuptake into presynaptic terminals..
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Deletion of the gamma-aminobutyric acid transporter 2 (GAT2 and SLC6A13) gene in mice leads to changes in liver and brain taurine contents.
These findings demonstrate that GABA uptake and release via GAT-1, GAT-3, and VGAT are initially established in the inner retina during the first postnatal week and that these systems then mature in the outer retina during the second postnatal week.
Neurotransmitter transporters including SLC6A6 and SLC6A13 mediate the uptake of 5-aminolevulinic acid (ALA) and can play roles in the enhancement of ALA-induced accumulation of protoporphyrin in cancerous cells.
Results suggest that gamma-aminobutyric acid transporter 2 GAT-2 is a high selectivity/low affinity transporter.
Terminates the action of GABA by its high affinity sodium-dependent reuptake into presynaptic terminals.
, gamma-aminobutyric acid (GABA-A) transporter 3
, sodium- and chloride-dependent GABA transporter 2
, sodium- and chloride-dependent GABA transporter 3
, solute carrier family 6 member 13
, Solute carrier family 6 member 13
, solute carrier family 6 (neurotransmitter transporter, GABA), member 13
, GABA transport protein
, GABA transporter 2