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SFTPD belongs to the homeobox family of genes. Additionally we are shipping Surfactant Associated Protein D Antibodies (18) and Surfactant Associated Protein D Proteins (8) and many more products for this protein.
Showing 3 out of 22 products:
Human SFTPD ELISA Kit for Sandwich ELISA - ABIN414528
Salman, Akpinar, Yigit, Gormus: Surfactant protein A and D in chronic rhinosinusitis with nasal polyposis and corticosteroid response. in American journal of rhinology & allergy 2012
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Rat (Rattus) SFTPD ELISA Kit for Sandwich ELISA - ABIN367579
Güzel, Güzel, Günaydin, Alaçam, Saliş, Sükrü Paksu, Murat, Gacar, Güvenç: The role of iNOS inhibitors on lung injury induced by gastrointestinal decontamination agents aspiration. in Journal of molecular histology 2012
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Rat (Rattus) SFTPD ELISA Kit for Sandwich ELISA - ABIN416223
Horie, Fukui, Endoh, Maru, Miyauchi, Shichiri, Fujita, Niki, Hagihara, Yoshida, Morimoto, Iwahashi: Comparison of acute oxidative stress on rat lung induced by nano and fine-scale, soluble and insoluble metal oxide particles: NiO and TiO2. in Inhalation toxicology 2012
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Mouse (Murine) SFTPD ELISA Kit for Sandwich ELISA - ABIN415784
Yu, Islam, Sikora, Dworski, Gurka, López-Vásquez, Liu, Kuebler, Levade, Zhang, Medin: Chronic lung injury and impaired pulmonary function in a mouse model of acid ceramidase deficiency. in American journal of physiology. Lung cellular and molecular physiology 2017
the SPA (show FASL ELISA Kits) and SPD levels in EBC were correlated with lung function, which contributed to COPD (show ARCN1 ELISA Kits) diagnosis.
Studied predictive value of surfactant protein D (SP-D) in lung cancer patients with interstitial lung disease induced by anticancer agents (ILD-AA). Results suggest that SP-D level change was a risk factor for mortality in patients with ILD-AA, and that SP-D might be a predictive prognostic biomarker of ILD-AA.
SP-D also delays FasL (show FASL ELISA Kits)-induced death of primary human T cells. SP-D delaying the progression of the extrinsic pathway of apoptosis could have important implications in regulating immune cell homeostasis at mucosal surfaces
findings suggest that these pulmonary markers could be useful to assess CAP severity and, especially YKL-40 (show CHI3L1 ELISA Kits) and CCL18 (show CCL18 ELISA Kits) by helping predict CAP caused by atypical pathogens
Trimeric SP-D wildtype recognized larger LPS inner core oligosaccharides with slightly enhanced affinity than smaller compounds suggesting the involvement of stabilizing secondary interactions.
rs2819096 in the surfactant protein D (SFTPD) gene was associated with a higher risk of COPD (show ARCN1 ELISA Kits) GOLD III + IV.
SP-D increases the formation of nuclear and membrane blebs. Inhibition of caspase-8 (show CASP8 ELISA Kits) confirms the effect of SP-D is unique to the caspase-8 (show CASP8 ELISA Kits) pathway.
Findings indicate serum pulmonary surfactant protein D (SP-D, SFTPD) level as a potential marker to estimate the efficacy of epidermal growth factor receptor (EGFR (show EGFR ELISA Kits))-tyrosine kinase (show TXK ELISA Kits) inhibitor (TKIs).
Patients with SP-D 11Thr/Thr (show TRH ELISA Kits) genotype were more susceptible to acute kidney injury (AKI). Compared with healthy controls, serum SP-D levels at day 1, 3 and 7 were significantly elevated in AKI patients.
This review intends to provide a current overview of the genetics, structure and extra-pulmonary functions of the surfactant collectin (show MBL2 ELISA Kits) proteins.
Elevation of SP-D is found in bronchoalveolar lavage fluid from inoculated lobes of calves infected with bovine adenovirus type 3.
The bSP (show KLK6 ELISA Kits)-D gene has 9 exons spanning about 10.5 kb on chromosome 28 at position q1.8. The 5'-upstream sequence has cis-regulatory elements. It has 2 (show HAS2 ELISA Kits) polymorphisms in the carbohydrate recognition domain (242 Glu (show DCTN1 ELISA Kits)/Val & 268 Ala/Gly).
The authors demonstrated that the number and position of glycosylation sites determine viral susceptibility to the neutralizing activity of porcine SP-D.
Data show that interactions of surfactant protein D (pSP-D) with influenza A virus mediated by the N-linked carbohydrate moiety in pSP-D depend on the terminal sialic acids present on this moiety.
The presence of SP-D in endothelial cells which is NO-, PI(3 (show PI3 ELISA Kits))K/Akt (show AKT1 ELISA Kits)- and Erk (show MAPK1 ELISA Kits)-dependent is demonstrated. It suggests a protective role of SP-D in these cells.
The importance of SP-D in innate immunity is underlined by its expression at the potential ports of entry of pathogens: lung, tongue, intestinal tract, thymus, skin, gall bladder, lacrimal gland, esophagus, stomach, kidney, liver, prostate and spleen.
Colitis caused a notable increase in Sftpd gene expression in the gallbladder, but not in the lung, via the activity of glucocorticoids produced in the liver.
SP-D deficiency reduces atherosclerosis in ApoE (show APOE ELISA Kits)-knockout mice by decreasing the accumulation and proliferation of macrophages and by reducing IL-6 (show IL6 ELISA Kits) levels systemically.
The data provide evidence SP-A (show SFTPA1 ELISA Kits) and SP-D attenuate S. aureus pneumonia severity resulting in decreased intestinal mucosal injury, apoptosis, and inflammation.
In triple KO mice, the HO-induced lung injury was associated with decreased surfactant protein (SP) A (show SFTPA1 ELISA Kits) and SPC (show SFTPC ELISA Kits) but not SPB and SPD expression.
Our study provides comprehensive information regarding spatial and temporal expression of collectins (SP-A, SP-D and MBL-A) and their cellular sources in murine testis. Positive regulation by testosterone and alteration in their level upon LPS challenge are clues relevant to suggest their involvement in testicular immune privilege.
Our data may promote further studies on the morphological development of the aging lung and the role of SP-D in this process.
The experimentally induced alterations were more severe in the SP-D(-/-) than in the WT mice, particularly with respect to the surfactant-storing lamellar bodies which were sometimes extremely enlarged in SP-D(-/-) mice.
these results demonstrate SP-D plays protective roles by inhibiting apoptosis and modulating NF-kappaB-mediated inflammation in CLP-induced acute pancreatic injury .
SP-A (show SFTPA1 ELISA Kits)/D double knock-out mice showed increased susceptibility to urinary tract infection
Human and murine data together indicate that SP-A, SP-D and MBL are synthesized in early gestational tissues, and may contribute to regulation of immune response at the feto-maternal interface during pregnancy.
Contributes to the lung's defense against inhaled microorganisms. May participate in the extracellular reorganization or turnover of pulmonary surfactant. Binds strongly maltose residues and to a lesser extent other alpha-glucosyl moieties.
, lung surfactant protein D
, pulmonary surfactant apoprotein
, pulmonary surfactant-associated protein D
, surfactant, pulmonary-associated protein D
, surfactant-associated protein, pulmonary 4
, pulmonary surfactant protein D
, surfactant associated protein D