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The protein encoded by TRIM33 is thought to be a transcriptional corepressor. Additionally we are shipping TRIM33 Proteins (6) and and many more products for this protein.
Showing 10 out of 117 products:
Human Monoclonal TRIM33 Primary Antibody for ICC, IF - ABIN261638
Tirard, Hsiao, Nikolov, Urlaub, Melchior, Brose: In vivo localization and identification of SUMOylated proteins in the brain of His6-HA-SUMO1 knock-in mice. in Proceedings of the National Academy of Sciences of the United States of America 2012
Human Monoclonal TRIM33 Primary Antibody for IF, IHC (p) - ABIN565640
Ding, Jin, Wang, Chen, Wu, Ai, Chen, Zhang, Liang, Laurence, Zhang, Datta, Zhang, Chen: Reduced expression of transcriptional intermediary factor 1 gamma promotes metastasis and indicates poor prognosis of hepatocellular carcinoma. in Hepatology (Baltimore, Md.) 2014
Tumors from paraneoplastic anti-TIF1gamma-positive cancer-associated myositis patients showed an increased number of genetic alterations, such as mutations and loss of heterozygosity, in TIF1 (show TRIM24 Antibodies) genes.
Data show that tripartite motif-containing protein 33 (TRIM33) silencing attenuates down-regulation of MYC (show MYC Antibodies) and TGF-beta (show TGFB1 Antibodies) signaling in response to bromodomain and extraterminal domain protein inhibitors (BETi).
suggest that SnoN (show SKIL Antibodies) suppresses TGF-betainduced epithelial-mesenchymal transition and invasion of bladder cancer cells in a TIF1gammadependent manner
The adenovirus E4-ORF3 protein functions as a SUMO E3 ligase for TIF-1gamma sumoylation and poly-SUMO chain elongation.
our findings reveal a new mechanism by which SOX2 (show SOX2 Antibodies)-mediated transcription repression of TIF1gamma promotes TGF-beta (show TGFB1 Antibodies)-induced epithelial-mesenchymal transition in non-small-cell lung cancer
our work indicates that TIF1gamma exerts its tumor-suppressive functions in part by promoting chromosomal stability.
Results show that TRIM33 is significantly downregulated in clear renal cell carcinoma (show MOK Antibodies) tissues which seems to correlate with pathologic stages and grades.
Tumour suppressor TRIM33 targets nuclear beta-catenin (show CTNNB1 Antibodies) degradation
Study suggests TRIM33 and NRAS (show NRAS Antibodies)-CSDE1 (show CDSE1 Antibodies) as candidate genes for autism, and may provide a novel insight into the etiology of autism
The dermatomyositis autoantigen TIF1gamma is markedly up-regulated during muscle regeneration in human and mouse muscle cells.
Ectodermin is a key switch in the control of TGF-beta (show TGFB1 Antibodies) gene responses during early embryonic development and cell proliferation.
Data show that tripartite motif 33 (TRIM33) mediates Wnt (show WNT2 Antibodies) signaling by directly regulating the expression of a specific subset of Wnt (show WNT2 Antibodies) target genes.
Trim33 is thus revealed as being essential for the navigation of macrophages and neutrophils towards developmental or inflammatory cues within vertebrate tissues.
Study reveals new important functions of TRIM33 in macrophage production and activation by LPS (show TLR4 Antibodies) and pinpoints TRIM33 as an important transcriptional actor of monocyte/macrophage mediated inflammation.
TRIM33 is necessary for osteoblast proliferation by regulating cell cycle and for differentiation via bone morphogenetic protein signaling pathway.
these results indicate that Trim33 deficiency leads to the expansion of a subset of myeloid cells characterizing the myelodysplastic/myeloproliferative neoplasm.
Trim33 altered expression of a small group of genes in the testis and the gene with the most significant increase was transcribed from an upstream RLTR10B.
TRIM33 deficiency results in high expression of Ifnb1 (show IFNB1 Antibodies) at late stages of macrophages activation.
The findings reveal an essential role for TRIM33 in preventing apoptosis in B lymphoblastic leukemia by interfering with enhancer-mediated Bim (show BCL2L11 Antibodies) activation.
This study demonistrated that Smad4 (show SMAD4 Antibodies) and Trim33 regulate neural stem cells in the developing cortex in mice.
The authors show that tif1gamma modulates the erythroid versus myeloid fate outcomes from HSCs by differentially controlling the levels of gata1 (show GATA1 Antibodies) and pu.1.
TIF1gamma couples the blood-specific transcriptional complex with Pol II (show 0 Antibodies) elongation machinery to promote the transcription elongation of erythroid genes by counteracting Pol II (show 0 Antibodies) pausing.
The protein encoded by this gene is thought to be a transcriptional corepressor. However, molecules that interact with this protein have not yet been identified. The protein is a member of the tripartite motif family. This motif includes three zinc-binding domains, a RING, a B-box type 1 and a B-box type 2, and a coiled-coil region. Three alternatively spliced transcript variants for this gene have been described, however, the full-length nature of one variant has not been determined.
tripartite motif-containing 33
, tripartite motif-containing 33 protein
, E3 ubiquitin-protein ligase TRIM33
, e3 ubiquitin-protein ligase TRIM33-like
, tripartite motif containing 33
, RET-fused gene 7 protein
, ectodermin homolog
, protein Rfg7
, transcriptional intermediary factor 1 gamma
, transcription intermediary factor 1-gamma
, tripartite motif-containing protein 33
, tripartite motif protein 33