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Human Polyclonal GCP2 Primary Antibody for WB - ABIN520075
Vaithilingam, Quayum, Joglekar, Jensen, Hardikar, Oberholzer, Guillemin, Tuch: Effect of alginate encapsulation on the cellular transcriptome of human islets. in Biomaterials 2011
Parenchymal polymorphonuclear myeloid-derived suppressor cell (PMN (show TBCE Antibodies)-MDSC), have a positive correlation with IL1a (show IL1A Antibodies), IL8 (show IL8 Antibodies), CXCL5 (show CXCL5 Antibodies), and Mip-1a (show CCL3 Antibodies), suggesting they may attract PMN (show TBCE Antibodies)-MDSC into the tumor
These data identify suppression of CXCL2 (show CXCL2 Antibodies) and CXCL5 (show CXCL5 Antibodies) chemoattractant expression by 11beta-HSD1 (show HSD11B1 Antibodies) as a novel mechanism with potential for regulation of neutrophil recruitment to the injured myocardium, and cardiac fibroblasts as a key site for intracellular glucocorticoid regeneration during acute inflammation following myocardial injury.
IL-17RA (show IL17RA Antibodies) regulates CXL-1 and 5 production in the lungs during the adaptive response.
STAT3 (show STAT3 Antibodies) is required for maximal OSM (show OSM Antibodies)-induced CXCL5 (show CXCL5 Antibodies) expression.
CXCL5 (show CXCL5 Antibodies) has a role in neutrophil recruitment in TH17-mediated glomerulonephritis
Since adaptive villus growth occurs despite impaired CXCL5 (show CXCL5 Antibodies) expression and enhanced angiogenesis, this suggests that the growth of new blood vessels is not needed for resection-induced mucosal surface area expansion following massive SBR (show NXF1 Antibodies).
CXCL5 (show CXCL5 Antibodies) regulates pulmonary responses to infection and plays a central role in conferring clock control of inflammation.
findings demonstrated that CXCL1 (show CXCL1 Antibodies) and CXCL5 (show CXCL5 Antibodies) are increased in circulation with onset of T2D, are produced by islets under stress, and synergistically affect islet function, suggesting that these chemokines participate in pathogenesis of T2D.
TLR2-induced epithelial-derived CXCL5 (show CXCL5 Antibodies) is critical for polymorphonuclear leukocyte-driven destructive inflammation in pulmonary tuberculosis.
Our data suggest that the differential regulation of the chemokine CXCL5 between osteoblasts and endothelial cells upon FGF2 treatment is involved in Hematopoietic stem cell mobilization from the osteoblast niche or bone marrow to peripheral blood.
There was underexpression of the majority of genes after sunitinib treatment. The lower expression levels of IGFBP1 (show IGFBPI Antibodies), CCL20 (show CCL20 Antibodies), CXCL6 and FGB (show FGB Antibodies) were confirmed by qRT-PCR in all cases. The downregulation of gene expression leads us to search for methylation as a mechanism of action of the tyrosine kinase (show TXK Antibodies) inhibitors
CXCL6 level is high in the serum of chronic hepatitis B patients.CXCL6 promotes human hepatocyte proliferation through the CXCR1-NFkappaB pathway and inhibits collagen I secretion by hepatic stellate cells.
The role of PITX2 (show PITX2 Antibodies) in glaucoma may be mediated partly by regulating the expression of CXCL6 and BBS5 (show BBS5 Antibodies) and thus affecting immune functions and intraocular pressure.
This is the first study to note that elevated systemic CCL5 (show CCL5 Antibodies) and CXCL6 were associated with moderate/severe lumbar disc degeneration. These chemokines may be systemic biomarkers for the diagnosis and monitoring of disc degeneration.
The neutrophil-recruiting chemokine (show CCL1 Antibodies) GCP-2/CXCL6 is expressed in cystic fibrosis (show S100A8 Antibodies) airways and retains its functional properties after binding to extracellular DNA.
HIF-1alpha (show HIF1A Antibodies) promotes HCC (show FAM126A Antibodies) progression and metastasis by upregulating CXCL6 transcription in HCC (show FAM126A Antibodies) cells.
Expression of the CXCL8 (show IL8 Antibodies), CXCL6 and CXCL1 (show CXCL1 Antibodies) genes are under the primary control of 1,25-dihydroxyvitamin D3 and its receptor.
Data suggest that expression of CXCL6 in trophoblasts is up-regulated during pregnancy development/placentation; CXCL6 expression inhibits trophoblast cell migration and invasion by down-regulating activity of MMP2 (matrix metalloproteinase 2 (show MMP2 Antibodies)).
Overexpression of GCP-2 (show FOLH1 Antibodies) in mesenchymal stem cells has the potential to enhance their angiogenic and survival properties.
Studies show that functional blocking of GCP-2 (show FOLH1 Antibodies) inhibits tumor growth and metastases.
mouse homolog is a member of the CXC chemokine family, is a neutrophil chemoattractant and is rapidly induced in response to muscle injury
C-X-C motif chemokine 5
, CXC chemokine LIX
, chemokine (C-X-C motif) ligand 6 (granulocyte chemotactic protein 2)
, cytokine LIX
, small-inducible cytokine B5
, granulocyte chemotactic protein-2
, small inducible cytokine B subfamily, member 5
, small inducible cytokine subfamily B, member 15
, C-X-C motif chemokine 6
, Small inducible cytokine subfamily B (Cys-X-Cys), member b
, chemokine alpha 3
, granulocyte chemotactic protein 2
, small inducible cytokine subfamily B (Cys-X-Cys), member 6 (granulocyte chemotactic protein 2)
, small-inducible cytokine B6
, chemokine (C-X-C motif) ligand 5
, inducible cytokine subfamily B (Cys-X-Cys), member 6