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anti-Human DDX17 Antibodies:
anti-Rat (Rattus) DDX17 Antibodies:
anti-Mouse (Murine) DDX17 Antibodies:
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Human Polyclonal DDX17 Primary Antibody for ICC, IF - ABIN152386
Jin, Chen, Di, Miron, Hou, Gao, Shao: Estrogen receptor (ER) beta or p53 attenuates ERalpha-mediated transcriptional activation on the BRCA2 promoter. in The Journal of biological chemistry 2008
Show all 3 Pubmed References
Mutant p53 protein (show TP53 Antibodies) (Mutp53) binds and sequesters RNA helicases p72/82 from microprocessor causing an attenuation of microRNAs (miRNAs) maturation.
DDX17 contributes to acquired gefitinib resistance through exportin (show XPO1 Antibodies)/importin-dependent cytoplasmic shuttling and activation of beta-catenin (show CTNNB1 Antibodies) in non-small lung cancer cells.
The miRNA biogenesis factors, DDX17 and KHSRP (show KHSRP Antibodies), regulate the protein level of Ago2 (show EIF2C2 Antibodies) in human cells.
DDX17 is a Sox2 (show SOX2 Antibodies)-binding protein in estrogen receptor (show ESR1 Antibodies)-positive breast cancer; in reporter responsive (RR) cells but not reporter unresponsive (RU) cells, DDX17 enhances the tumorigenic and stem-like features of Sox2 (show SOX2 Antibodies) by promoting its binding to its target genes
Overexpression of p72 decreased Beclin1 (show BECN1 Antibodies) expression partially by increasing miR (show MLXIP Antibodies)-34-5p and miR (show MLXIP Antibodies)-5195-3p expression in glioma cells.
Systematic Determination of Human Cyclin Dependent Kinase (show CDK1 Antibodies) (CDK)-9 (show CDK9 Antibodies) Interactome Identifies Novel Functions in RNA Splicing Mediated by the DDX5 (show DDX5 Antibodies) and DDX17 RNA Helicases
Downregulation of DDX5 (show DDX5 Antibodies) and DDX17 protein expression during myogenesis and epithelial-to-mesenchymal transdifferentiation contributes to the switching of splicing programs during these processes.
Depletion of DDX17 but not the related helicase DDX5 (show DDX5 Antibodies) increased Rift Valley fever virus replication in human cells.
DDX17 promotes the production of HIV-1 infectious particles by modulating HIV-1 RNA metabolism.
Data indicate that transcriptional coregulator ddx5 (show DDX5 Antibodies)/ddx17 RNA helicases can simultaneously regulate the transcriptional activity and alternative splicing of NFAT5 (show NFAT5 Antibodies) transcription factor.
Data show that p72/DDX17 specifically interacts with the miR (show MLXIP Antibodies)-132 loop sequence and influences the relative ratio of the mature mice miR (show MLXIP Antibodies)-212/132 miRNAs.
DEAD box proteins, characterized by the conserved motif Asp-Glu-Ala-Asp (DEAD), are putative RNA helicases. They are implicated in a number of cellular processes involving alteration of RNA secondary structure, such as translation initiation, nuclear and mitochondrial splicing, and ribosome and splicesosome assembly. Based on their distribution patterns, some members of this family are believed to be involved in embryogenesis, spermatogenesis, and cellular growth and division. This gene encodes a DEAD box protein, which is an ATPase activated by a variety of RNA species, but not by dsDNA. This protein, and that encoded by DDX5 gene, are more closely related to each other than to any other member of the DEAD box family. This gene can encode multiple isoforms due to both alternative splicing and the use of alternative translation initiation codons, including a non-AUG (CUG) start codon.
DEAD (Asp-Glu-Ala-Asp) box polypeptide 17
, DEAD box polypeptide 17
, DEAD box protein p72
, DEAD/H (Asp-Glu-Ala-Asp/His) box polypeptide 17 (72kD)
, RNA-dependent helicase p72
, probable ATP-dependent RNA helicase DDX17
, DEAD (Asp-Glu-Ala-Asp) box polypeptide 46
, DEAD box protein 17