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anti-Human Growth Hormone 1 Antibodies:
anti-Mouse (Murine) Growth Hormone 1 Antibodies:
anti-Rat (Rattus) Growth Hormone 1 Antibodies:
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Human Polyclonal Growth Hormone 1 Primary Antibody for - ABIN285499
Staloch, Divine, Witten, Simon: C/EBP and Cdx family factors regulate liver fatty acid binding protein transgene expression in the small intestinal epithelium. in Biochimica et biophysica acta 2005
Human Monoclonal Growth Hormone 1 Primary Antibody for ICC, IF - ABIN268996
Thompson, Seethala, Müller: Ectopic sphenoid sinus pituitary adenoma (ESSPA) with normal anterior pituitary gland: a clinicopathologic and immunophenotypic study of 32 cases with a comprehensive review of the english literature. in Head and neck pathology 2012
Human Monoclonal Growth Hormone 1 Primary Antibody for FACS, IF - ABIN5578783
Niall, Hogan, Sauer, Rosenblum, Greenwood: Sequences of pituitary and placental lactogenic and growth hormones: evolution from a primordial peptide by gene reduplication. in Proceedings of the National Academy of Sciences of the United States of America 1971
Rat (Rattus) Polyclonal Growth Hormone 1 Primary Antibody for IHC (fro), WB - ABIN2473905
Ailus, Melamies, Tuomi, Palosuo, Aho: IgM-rheumatoid factors measured by ELISA in non-rheumatoid sera: comparison of human and rabbit Fc fragments as antigens. in British journal of rheumatology 1992
Show all 2 Pubmed References
GH1 and GHRHR screening revealed eleven variations in 24 (21%) patients with isolated growth hormone deficiency of which, four were novel deleterious, one novel non-pathogenic and six reported changes.
The results suggest that GH regulates energy metabolism directly in myocytes and that UCP2 (show UCP2 Antibodies) participates in the signal transduction pathway that functions downstream of the GHR (show GHR Antibodies)/JAK (show JAK3 Antibodies)/STAT (show STAT1 Antibodies).
These results implicate TIMP3 (show TIMP3 Antibodies) as a modulator of cell surface GHR (show GHR Antibodies) abundance and the ability of GH to promote cellular signaling.
Children with GH excess underwent medical treatment with lanreotide and a minimum clinical/biochemical follow up of 2 years is reported. The present study demonstrates that GH excess should be considered as a relative frequent endocrine manifestation in NF1 (show NF1 Antibodies) patients, similarly to central precocious puberty
Thus, GHRH (show GHRH Antibodies) analogs of the Miami series powerfully suppress tumor growth, but have only a weak endocrine GH inhibitory activity. The suppression of tumor growth could be induced in part by the downregulation of GHRH (show GHRH Antibodies) receptors levels.
The intrinsic amyloidogenicity of growth hormone, in the presence of contaminating prion protein (show PRNP Antibodies) (and perhaps prolactin (show PRL Antibodies) as well) and amyloid-beta contained in some cadavers' pituitaries, may have led to the observed co-occurring of Creutzfeldt-Jakob disease and Alzheimer's disease.
To our knowledge, c.-223C>T is the first homozygous point mutation in the GH1 promoter that leads to short stature due to idiopathic growth hormone deficiency.
Data show that the recombinant protein produced by the plasmid-free E coli strain was purified and characterized to be human growth hormone (hGH).
Our results suggest that the known protective effect of GH signaling deficiency on neoplastic tissue growth is mediated, at least partially, by regulating p53 (show TP53 Antibodies) expression
Gene polymorphism of leptin (loci rs7799039) and leptin receptor (loci rs1137101) are correlated with Growth hormone deficiency susceptibility.
These results indicate that up-regulation of GH in the lungs of DJ-1 (show PARK7 Antibodies) KO mice may enhance the malignancy of B16F10 cells and nodule formation in pulmonary metastasis of melanoma.
Confirmation of the impairment of GH-IGF-1 (show IGF1 Antibodies) release in hyperphagic MC4R (show MC4R Antibodies) KO mice suggests a role for insulin (show INS Antibodies) in regulating both the release of GH, but also in mediating growth during periods of physiologically suppressed GH-IGF-1 (show IGF1 Antibodies) levels
When charged with hypoxia-ischemia, mutant brains with deleted IGF-1 (show IGF1 Antibodies) receptor were broadly protected from cell damage, neuroinflammation and cerebral edema.
Thiol-disulfide exchange reactions in hGH and related model peptides were influenced by higher order structure, by the size of the thiol reactant and by an Arg residue adjacent to Cys (show DNAJC5 Antibodies) in the thiol reactant.
Growth hormone deficient mice exhibited renal hypertrophy in tubular epithelial cells.
observations demonstrate that germline loss of ghrelin-O-acyltransferase alters Growth Hormone release and patterning
alter miR (show MLXIP Antibodies)-19b concentrations in hematopoietic stem cells (HSCs) and affect HSC (show FUT1 Antibodies) migration
Moderate elevations in circulating GH and IGF-I (show IGF1 Antibodies) can directly increase basal insulin (show INS Antibodies) secretion without impacting beta-cell mass, independent of changes in whole body insulin (show INS Antibodies) sensitivity and hyperlipidemia.
Acylated ghrelin (show GHRL Antibodies) is not required for the surge in pituitary growth hormone observed in pregnant mice.
STAT5 (show STAT5A Antibodies) signaling is increased in the liver in GH-transgenic mice during the growth period, with a balance between positive and negative effectors resulting in accelerated but controlled growth.
bGH mice had increased body weight and decreased percent fat mass. Serum FGF21 (show FGF21 Antibodies) levels were significantly elevated in bGH mice. Expression of Fgf21 (show FGF21 Antibodies), Fgfr1 (show FGFR1 Antibodies), and Klb (show KLB Antibodies) mRNA in white AT and liver were downregulated or unchanged in bGH mice.
GH/HpaII locus as candidate marker for body weight in cattle rather than MSTN (show MSTN Antibodies)/DraI.
An SNP GH4.1 in a growth hormone gene was significantly related to a weaning weight.
The association of IGF1 (show IGF1 Antibodies), GH, and PIT1 (show POU1F1 Antibodies) markers with growth and reproductive traits were assessed.
The total frequencies for the combined genotypes for the bGH and RORC (show RORC Antibodies) genes, which provide for superior meat quality and carcass weight, in the populations of Kazakh white-headed cattle
Whereas only moderate structural changes were observed with up-regulation of GH/IGF-I (show IGF1 Antibodies) axis, disruption of the GH receptor (show GHR Antibodies) had pronounced effects upon tendon ultra-structure.
this study identifies a biochemical mechanism for the regulation of SCFAs on bovine GH and PRL (show PRL Antibodies) gene transcription in dairy cow anterior pituitary cells
homozygote genotypes of GH (LL) and beta-LG (AA) were superior compared to heterozygote genotypes, whereas, the heterozygote genotype of Pit-1 (show POU1F1 Antibodies) gene (AB) was desirable
one GH1 SNP, GH33, was significantly associated with milk protein (show CSN2 Antibodies) yield in the second lactation. Several GH1 SNPs were significantly associated with fertility.
GH and IGF-I (show IGF1 Antibodies) genotypes had no substantial effect on productive parameters, though IGF-I (show IGF1 Antibodies) genotype affected calving-first service interval in primiparous cows. However, the genotypes do alter the endocrine/metabolic profiles of the transition dairy cow.
Data indicate that the dual effects of cortistatin (show CORT Antibodies) on growth hormone release parallel those of somatostatin (show SST Antibodies) and are probably mediated by the same receptor(s) and signaling pathway(s) for both peptides.
ghrelin increased growth hormone secretion but not growth hormone synthesis by ovarian follicles
Plasma levels of growth hormone (as well as glucose, insulin (show INS Antibodies), and leptin (show LEP Antibodies)) are highly correlated with the duration of winter anovulatory phase.
Expression of PRL (show PRL Antibodies) and GH in the guinea pig is prominent in the anterior pituitary, similar to known expression patterns of PRL (show PRL Antibodies) and GH for other species.
Reproductive tests showed that double transgenic males did not differ from non-transgenics. It is possible that GHR (show GHR Antibodies) excess in the muscle tissues of double transgenics may have contributed to lower circulating GH levels and thus reduced the negative effects of this hormone with respect to reproduction.
that concomitant overexpression of GH and GHR (show GHR Antibodies) resulted in a strong decrease of the somatotrophic axis intracellular signaling by diminishing its principal transcription factor signal transducer and activator of transcription 5.1 (show STAT5B Antibodies).
Effects of somatotrophic axis (GH/GHR (show GHR Antibodies)) double transgenesis on structural and molecular aspects of the zebrafish immune system
The zebrafish gh1 mutant, vizzini, exhibits decreased somatic growth, increased adipose tissue accumulation, and disrupted adipose plasticity after nutrient deprivation.
tbx5 (show TBX5 Antibodies) knockdown causes a pseudo GH deficiency in zebrafish during early embryonic stages, and supplementation of exogenous GH can partially restore dysmorphogenesis, apoptosis, cell growth inhibition, and abnormal cardiomyogenesis
Findings show similar regulatory growth hormone (GH) and insulin-like growth factor 1 (IGF-1 (show IGF1 Antibodies)) responses in both Atlantic salmon and rainbow trout.
The protein encoded by this gene is a member of the somatotropin/prolactin family of hormones which play an important role in growth control. The gene, along with four other related genes, is located at the growth hormone locus on chromosome 17 where they are interspersed in the same transcriptional orientation\; an arrangement which is thought to have evolved by a series of gene duplications. The five genes share a remarkably high degree of sequence identity. Alternative splicing generates additional isoforms of each of the five growth hormones, leading to further diversity and potential for specialization. This particular family member is expressed in the pituitary but not in placental tissue as is the case for the other four genes in the growth hormone locus. Mutations in or deletions of the gene lead to growth hormone deficiency and short stature.
pituitary growth hormone
, growth hormone 12
, growth hormone
, growth hormone 1
, neural retina growth hormone
, gnRH receptor
, gonadotropin-releasing hormone receptor
, chorionic somatommamotropin hormone 4
, Pituitary growth hormone
, growth hormone I
, Growth hormone
, growth hormone type a long isoform
, growth hormone type a short isoform
, growth hormone protein
, Growth hormone 1
, growth hormone B3
, growth hormone type 1