CLN6 Protein (Myc-DYKDDDDK Tag)
CLN6
Origin: Human
Host: HEK-293 Cells
Recombinant
> 80 % as determined by SDS-PAGE and Coomassie blue staining
AbP, STD
1 image
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- Target See all CLN6 Proteins
- CLN6 (Ceroid-Lipofuscinosis, Neuronal 6, Late Infantile, Variant (CLN6))
- Protein Type
- Recombinant
- Origin
- Human
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Source
- HEK-293 Cells
- Purification tag / Conjugate
- This CLN6 protein is labelled with Myc-DYKDDDDK Tag.
- Application
- Antibody Production (AbP), Standard (STD)
- Characteristics
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- Recombinant human CLN6 protein expressed in HEK293 cells.
- Produced with end-sequenced ORF clone
- Purity
- > 80 % as determined by SDS-PAGE and Coomassie blue staining
- Top Product
- Discover our top product CLN6 Protein
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Ceroid-Lipofuscinosis, Neuronal 6, Late Infantile, Variant (CLN6) (AA 1-311) protein (GST tag)
CLN6 Origin: Human Host: Wheat germ Recombinant ELISA, WB, AA, AP
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- Application Notes
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Recombinant human proteins can be used for:
Native antigens for optimized antibody production
Positive controls in ELISA and other antibody assays - Comment
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The tag is located at the C-terminal.
- Restrictions
- For Research Use only
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- Concentration
- 50 μg/mL
- Buffer
- 25 mM Tris.HCl, pH 7.3, 100 mM glycine, 10 % glycerol.
- Storage
- -80 °C
- Storage Comment
- Store at -80°C. Thaw on ice, aliquot to individual single-use tubes, and then re-freeze immediately. Only 2-3 freeze thaw cycles are recommended.
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- Target
- CLN6 (Ceroid-Lipofuscinosis, Neuronal 6, Late Infantile, Variant (CLN6))
- Alternative Name
- Cln6 (CLN6 Products)
- Synonyms
- 1810065L06Rik Protein, AW743417 Protein, D9Bwg1455e Protein, nclf Protein, CLN4A Protein, HsT18960 Protein, cln6 Protein, zgc:103565 Protein, ceroid-lipofuscinosis, neuronal 6 Protein, CLN6, transmembrane ER protein Protein, CLN6, transmembrane ER protein S homeolog Protein, ceroid-lipofuscinosis, neuronal 6, late infantile, variant Protein, CLN6, transmembrane ER protein a Protein, Cln6 Protein, CLN6 Protein, cln6.S Protein, cln6a Protein
- Background
- This gene is one of eight which have been associated with neuronal ceroid lipofuscinoses (NCL). Also referred to as Batten disease, NCL comprises a class of autosomal recessive, neurodegenerative disorders affecting children. The genes responsible likely encode proteins involved in the degradation of post-translationally modified proteins in lysosomes. The primary defect in NCL disorders is thought to be associated with lysosomal storage function.
- Molecular Weight
- 35.7 kDa
- NCBI Accession
- NP_060352
- Pathways
- Glycosaminoglycan Metabolic Process
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