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anti-Human NOX1 Antibodies:
anti-Rat (Rattus) NOX1 Antibodies:
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Human Polyclonal NOX1 Primary Antibody for ICC, IF - ABIN441545
Balasubramaniyan, Wright, Sharma, Davies, Sharifi, Habtesion, Mookerjee, Jalan: Ammonia reduction with ornithine phenylacetate restores brain eNOS activity via the DDAH-ADMA pathway in bile duct-ligated cirrhotic rats. in American journal of physiology. Gastrointestinal and liver physiology 2011
Show all 11 Pubmed References
Human Polyclonal NOX1 Primary Antibody for WB - ABIN4893182
Quintela, Jiménez, Gómez-Guzmán, Zarzuelo, Galindo, Sánchez, Vargas, Cogolludo, Tamargo, Pérez-Vizcaíno, Duarte: Activation of peroxisome proliferator-activated receptor-β/-δ (PPARβ/δ) prevents endothelial dysfunction in type 1 diabetic rats. in Free radical biology & medicine 2012
Show all 3 Pubmed References
Mouse (Murine) Polyclonal NOX1 Primary Antibody for IHC (p), WB - ABIN3044251
Wang, Wang, Liu, Wang, Zhao, Wang, Yin: Cordyceps sinensis polysaccharide inhibits PDGF-BB-induced inflammation and ROS production in human mesangial cells. in Carbohydrate polymers 2015
Show all 2 Pubmed References
Human Polyclonal NOX1 Primary Antibody for IF (p), IHC (p) - ABIN702580
Kashiwabara, Ambe, Nakagawa, Watanabe: Immunohistochemical localization of Nox in mouse circumvallate papillae. in Tissue & cell 2015
Show all 2 Pubmed References
Cow (Bovine) Polyclonal NOX1 Primary Antibody for WB - ABIN2782754
Kuwano, Kawahara, Yamamoto, Teshima-Kondo, Tominaga, Masuda, Kishi, Morita, Rokutan: Interferon-gamma activates transcription of NADPH oxidase 1 gene and upregulates production of superoxide anion by human large intestinal epithelial cells. in American journal of physiology. Cell physiology 2006
Show all 2 Pubmed References
Human Polyclonal NOX1 Primary Antibody for IHC (p), WB - ABIN3044132
Gu, Gong, Zhang, Dong, Zhao, Burczynski, Wang, Sun, Zhu, Han, Wang, Li: Regulation of transforming growth factor beta 1 gene expression by dihydropteridine reductase in kidney 293T cells. in Biochemistry and cell biology = Biochimie et biologie cellulaire 2013
Show all 2 Pubmed References
Human Polyclonal NOX1 Primary Antibody for ICC, IF - ABIN4335324
Fan, Hsiung, Cheng, Tzanakakis: Facile engineering of xeno-free microcarriers for the scalable cultivation of human pluripotent stem cells in stirred suspension. in Tissue engineering. Part A 2014
NOX activation might have a role in regulation of lymphocytic activity in patients with idiopathic nephrotic syndrome through the impairment of PDGF (show PDGFA Antibodies) mitogenic function and might contribute toward pathogenesis of nephrotic syndrome.
The SUMO1 (show SUMO1 Antibodies)/UBC9 (show UBE2I Antibodies) axis may regulate Nox1mediated diabetic retinopathy by inhibiting reactive oxygen species generation and apoptosis.
Results show that the thrombospondin 1 (TSP1 (show THBS1 Antibodies)) and its receptor CD47 (CD47 (show CD47 Antibodies)) axis selectively regulates NADPH oxidase 1 (Nox1) in the regulation of endothelial senescence and suggest potential targets for controlling the aging process at the molecular level.
in CAD (show CAD Antibodies), both mitochondria and NADPH oxidase contribute to flow-induced vasodilation through a redox mechanism in visceral arterioles.
NADPH oxidase-mediated redox signaling is important in the detrimental effect of C-reactive protein (show CRP Antibodies) on pancreatic insulin (show INS Antibodies) secretion.
S340E mutation enhances Nox1 activation (Kaito et al., 2014), the present study suggests that betaPix (show ARHGEF7 Antibodies) can also play an inhibitory role in O2(-) production, depending on the sites of phosphorylation.
the anti-proliferative and pro-apoptotic effect of cambogin on breast adenocarcinoma is mediated via inducing NOX1-dependent ROS (show ROS1 Antibodies) production and the dissociation of ASK1 (show MAP3K5 Antibodies) and Trx1 (show MLL Antibodies)
Transcriptional regulation of NOX genes expression in human breast adenocarcinoma cells is modulated by adaptor protein CIN85 (show SH3KBP1 Antibodies).
the transition-state substrate analogue inhibitor of Prdx6 phospholipase A2 activity (MJ-33) was shown to suppress Nox1 activity, suggesting Nox1 activity is regulated by the phospholipase activity of Prdx6. Finally, wild type Prdx6, but not lipase or peroxidase mutant forms, supports Nox1-mediated cell migration in the HCT-116 colon epithelial cell model of wound closure
Cells redox environment mediated by NOX1 isozymes activation down-regulates BRCA1 expression and promotes DNA homologous recombination repair in cancer.
these data demonstrate that altered zinc distribution leading to accumulation of zinc in the mitochondria increases mitochondrial ROS (show ROS1 Antibodies) production causing NF-kappaB (show NFKB1 Antibodies) activation which in turn upregulates Nox1 expression inducing senescence of vascular smooth muscle cells
our data identify EBP50 (show SLC9A3R1 Antibodies) as a previously unidentified regulator of Nox1 and support that it promotes Nox1 activity by binding p47(phox) This interaction is pivotal for agonist-induced smooth muscle ROS (show ROS1 Antibodies), hypertrophy, and vasoconstriction and has implications for ROS (show ROS1 Antibodies)-mediated physiological and pathophysiological processes.
We examined the pattern of NOX expression in spinal cords of patients and mouse models of ALS and analyzed the impact of genetic deletion of the NOX1 and 2 isoforms as well as pharmacological NOX inhibition in the SOD1 (show SOD1 Antibodies)(G93A) ALS mouse model.In contrast to previous publications, survival of SOD1 (show SOD1 Antibodies)(G93A) mice was not modified upon breeding with constitutive NOX1 and NOX2 (show CYBB Antibodies) deficient mice.
Both Nox1 and Duox2 (show DUOX1 Antibodies) induce exfoliation of crypt epithelium, but only Nox1 induces apoptosis. NOX1 and DUOX2 (show DUOX1 Antibodies) may be potential therapeutic targets for treating ileocolitis in human patients suffering inflammatory bowel disease (IBD).
we used the Ang II (show AGT Antibodies) infused hph-1 (show EGLN2 Antibodies) mice to examine the roles of NOX isoforms in the development of AAA (show AAAS Antibodies). We generated double mutants of hph-1 (show EGLN2 Antibodies)-NOX1, hph-1 (show EGLN2 Antibodies)-NOX2 (show CYBB Antibodies), hph-1 (show EGLN2 Antibodies)-p47phox, and hph-1 (show EGLN2 Antibodies)-NOX4 (show NOX4 Antibodies)
C-kit (show KIT Antibodies)-positive hematopoietic stem/progenitor cells expressed significantly higher of Nox1 and catalase (show CAT Antibodies), but less of lactoperoxidase (show LPO Antibodies) than in matured mononuclear cells.
Nox1 deletion reduces oxidant load and restores microvascular health in obese mice.
Data suggests that ROS (show ROS1 Antibodies) produced during primitive endoderm differentiation is dependent in part on increased NOX1 and NOX4 (show NOX4 Antibodies) levels, which is under the control of GATA6 (show GATA6 Antibodies). Furthermore, these results suggest that the combined activity of multiple NOX proteins is necessary for the differentiation of F9 cells to primitive endoderm.
Gp91phox (show CYBB Antibodies) NADPH oxidase modulates litter size by up-regulating mucin1 (show MUC1 Antibodies) expression in the uterus of mice.
NADPH oxidase plays an important role in proMMP-2 expression and activation and MMP-2 (show MMP2 Antibodies) mediated SMC (show DYM Antibodies) proliferation occurs through the involvement of Spm (show NPC1 Antibodies)-Cer (show CBLN1 Antibodies)-S1P (show MBTPS1 Antibodies) signaling axis under ANG II (show AGT Antibodies) stimulation of PASMCs
Differential Roles of Protein Complexes NOX1-NOXO1 and NOX2-p47phox in Mediating Endothelial Redox Responses to Oscillatory and Unidirectional Laminar Shear Stress.
The current study was designed to determine mechanisms underlying 20-hydroxyeicosatetraenoic acid -stimulated nitric oxide (NO) release, and particularly the role of NADPH oxidase, reactive oxygen species, and PI3-kinase (show PIK3CA Antibodies) in stimulated NO release.
NEP (show MME Antibodies) expression is down-regulated in vascular endothelial cells by physiological laminar shear, possibly via a mechanotransduction mechanism involving NADPH oxidase-induced reactive oxygen species production.
The nox1 expression in zebrafish during early nervous system development from 12 to 48 hours post fertilization.
Over inhibition of the NADPH oxidase by the NADPH (show NQO1 Antibodies) Inhibitor DPI (show DSP Antibodies) may reduce the cell even the tissue in the progress of healing after the injury, in zebrafish liver cells.
This gene encodes a member of the NADPH oxidase family of enzymes responsible for the catalytic one-electron transfer of oxygen to generate superoxide or hydrogen peroxide. Alternatively spliced transcript variants encoding multiple isoforms have been observed for this gene.
NADH/NADPH mitogenic oxidase subunit P65-MOX
, NADPH oxidase homolog-1
, mitogenic oxidase (pyridine nucleotide-dependent superoxide-generating)
, mitogenic oxidase 1
, NADH/NADPH mitogenic oxidase subunit p65-mox
, GP91 phox homolog
, NADPH oxidase 1 alpha
, NADPH oxidase-1
, predicted NADPH oxidase-1
, NADPH oxidase 1