BAD antibody (pTyr113) (FITC)

Catalog No. ABIN1967594

The Rabbit Polyclonal anti-BAD antibody is suitable to detect BAD in samples from Mouse. It has been validated for ELISA and DB.

Quick Overview for BAD antibody (pTyr113) (FITC) (ABIN1967594)

Target: BAD (BCL2-Associated Agonist of Cell Death (BAD))

Reactivity: Mouse

Host: Rabbit

Clonality: Polyclonal

Conjugate: This BAD antibody is conjugated to FITC

Application: ELISA, Dot Blot (DB)

Product Details anti-BAD Antibody (FITC)

Binding Specificity: pTyr113

Specificity: This mouse BAD Antibody is generated from rabbits immunized with a KLH conjugated synthetic phosphopeptide corresponding to amino acid residues surrounding Y113 of mouse BAD.

Purification: Affinity purified

Isotype: IgG

Application Details

Application Notes: Approved: DB, ELISA

Usage: The applications listed have been tested for the unconjugated form of this product. Other forms have not been tested.

Comment:

Target Species of Antibody: Mouse

Restrictions: For Research Use only

Handling

Format: Liquid

Concentration: Lot specific

Buffer: PBS, no preservatives added

Preservative: Without preservative

Handling Advice: Aliquot to avoid repeated freezing and thawing.

Storage: 4 °C,-20 °C

Storage Comment: Short term: store at 4°C. Long term: aliquot and store -20°C for up to 6 months. Avoid freeze-thaw cycles. Protect from light.

Expiry Date: 6 months

Target Details for BAD

Target: BAD (BCL2-Associated Agonist of Cell Death (BAD))

Alternative Name: BAD

Background: Name/Gene ID: BAD
Family: Apoptosis

Synonyms: BAD, BBC2, Bcl-2-like protein 8, BCL2-binding component 6, BCL2L8, BBC6, BCL-X/BCL-2 binding protein, Bcl-2-binding component 6, Bcl2 antagonist of cell death, BCL2-binding protein, Bcl2-L-8

Gene ID: 572

Pathways: MAPK Signaling, PI3K-Akt Signaling, RTK Signaling, Apoptosis, Fc-epsilon Receptor Signaling Pathway, Positive Regulation of Peptide Hormone Secretion, Carbohydrate Homeostasis, Positive Regulation of Endopeptidase Activity, Regulation of Carbohydrate Metabolic Process, Hepatitis C, CXCR4-mediated Signaling Events